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Journal of Molecular and Cellular Cardiology|December 17, 2017
Absence of synemin in mice causes structural and functional abnormalities in heartKarla P García-Pelagio, Ling Chen, Humberto C Joca, et al.
Current Issues in Molecular Biology|July 28, 2023
Structural Analysis of Mitochondria in Cardiomyocytes: Insights into Bioenergetics and Membrane RemodelingRaquel A Adams, Zheng Liu, Chongere Hsieh, et al.
The Journal of Biological Chemistry|November 3, 2010
Novel function of cardiac protein kinase D1 as a dynamic regulator of Ca2+ sensitivity of contractionMariah H Goodall, Robert D Wardlow, Rebecca R Goldblum, et al.
The Journal of Clinical Investigation|October 14, 1998
Transgenic expression of replication-restricted enteroviral genomes in heart muscle induces defective excitation-contraction coupling and dilated cardiomyopathyR Wessely, K Klingel, L F Santana, et al.
Cell|June 28, 2011
Golgi export of the Kir2.1 channel is driven by a trafficking signal located within its tertiary structureDonghui Ma, Tarvinder Kaur Taneja, Brian M Hagen, et al.
Annual International Conference of the IEEE Engineering in Medicine and Biology Society. IEEE Engineering in Medicine and Biology Society. Annual International Conference|January 19, 2012
Stochastic simulation of cardiac ventricular myocyte calcium dynamics and wavesHoang-Trong Minh Tuan, George S B Williams, Aristide C Chikando, et al.
The Journal of Biological Chemistry|October 29, 2002
Protein kinase A phosphorylation of the cardiac calcium release channel (ryanodine receptor) in normal and failing hearts. Role of phosphatases and response to isoproterenolSteven Reiken, Marta Gaburjakova, Silvia Guatimosim, et al.
JCI Insight|October 26, 2016
IP<sub>3</sub> receptors regulate vascular smooth muscle contractility and hypertensionQingsong Lin, Guiling Zhao, Xi Fang, et al.
Circulation Research|January 5, 2008
AKAP150 is required for stuttering persistent Ca2+ sparklets and angiotensin II-induced hypertensionManuel F Navedo, Madeline Nieves-Cintrón, Gregory C Amberg, et al.
American Journal of Physiology. Heart and Circulatory Physiology|September 18, 2002
Defective intracellular Ca(2+) signaling contributes to cardiomyopathy in Type 1 diabetic ratsKin M Choi, Yan Zhong, Brian D Hoit, et al.
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