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Lishan Su

Showing results (131-140 of 153) with videos related to

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The Journal of Clinical Investigation|December 13, 2016
Blocking type I interferon signaling enhances T cell recovery and reduces HIV-1 reservoirsLiang Cheng, Jianping Ma, Jingyun Li, et al.
Plos Pathogens|January 6, 2018
Infection and depletion of CD4+ group-1 innate lymphoid cells by HIV-1 via type-I interferon pathwayJuanjuan Zhao, Liang Cheng, Hongbo Wang, et al.
Cell Reports|December 12, 2019
Type-I-IFN-Stimulated Gene TRIM5γ Inhibits HBV Replication by Promoting HBx DegradationGuangyun Tan, Zhaohong Yi, Hongxiao Song, et al.
The Journal of Infectious Diseases|May 20, 2011
B and T lymphocyte attenuator down-regulation by HIV-1 depends on type I interferon and contributes to T-cell hyperactivationZheng Zhang, Xiangsheng Xu, Jiyun Lu, et al.
The Journal of Clinical Investigation|June 26, 2010
Mitigation of hematologic radiation toxicity in mice through pharmacological quiescence induced by CDK4/6 inhibitionSøren M Johnson, Chad D Torrice, Jessica F Bell, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 8, 2019
Hepatitis B Virus-Induced Imbalance of Inflammatory and Antiviral Signaling by Differential Phosphorylation of STAT1 in Human MonocytesHongxiao Song, Guangyun Tan, Yang Yang, et al.
Scientific Reports|December 4, 2014
Interferon-inducible cholesterol-25-hydroxylase inhibits hepatitis C virus replication via distinct mechanismsYongzhi Chen, Shanshan Wang, Zhaohong Yi, et al.
Immunity|January 1, 2013
Interferon-inducible cholesterol-25-hydroxylase broadly inhibits viral entry by production of 25-hydroxycholesterolSu-Yang Liu, Roghiyh Aliyari, Kelechi Chikere, et al.
Nature Communications|January 10, 2017
Hepatitis C virus has a genetically determined lymphotropism through co-receptor B7.2Chia-Lin Chen, Jeffrey Y Huang, Chun-Hsiang Wang, et al.
The Journal of Biological Chemistry|October 6, 2005
The CATERPILLER protein monarch-1 is an antagonist of toll-like receptor-, tumor necrosis factor alpha-, and Mycobacterium tuberculosis-induced pro-inflammatory signalsKristi L Williams, John D Lich, Joseph A Duncan, et al.
Pageof 16

Showing results (131-140 of 153) with videos related to

Sort By:
Pageof 16
The Journal of Clinical Investigation|December 13, 2016
Blocking type I interferon signaling enhances T cell recovery and reduces HIV-1 reservoirsLiang Cheng, Jianping Ma, Jingyun Li, et al.
Plos Pathogens|January 6, 2018
Infection and depletion of CD4+ group-1 innate lymphoid cells by HIV-1 via type-I interferon pathwayJuanjuan Zhao, Liang Cheng, Hongbo Wang, et al.
Cell Reports|December 12, 2019
Type-I-IFN-Stimulated Gene TRIM5γ Inhibits HBV Replication by Promoting HBx DegradationGuangyun Tan, Zhaohong Yi, Hongxiao Song, et al.
The Journal of Infectious Diseases|May 20, 2011
B and T lymphocyte attenuator down-regulation by HIV-1 depends on type I interferon and contributes to T-cell hyperactivationZheng Zhang, Xiangsheng Xu, Jiyun Lu, et al.
The Journal of Clinical Investigation|June 26, 2010
Mitigation of hematologic radiation toxicity in mice through pharmacological quiescence induced by CDK4/6 inhibitionSøren M Johnson, Chad D Torrice, Jessica F Bell, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 8, 2019
Hepatitis B Virus-Induced Imbalance of Inflammatory and Antiviral Signaling by Differential Phosphorylation of STAT1 in Human MonocytesHongxiao Song, Guangyun Tan, Yang Yang, et al.
Scientific Reports|December 4, 2014
Interferon-inducible cholesterol-25-hydroxylase inhibits hepatitis C virus replication via distinct mechanismsYongzhi Chen, Shanshan Wang, Zhaohong Yi, et al.
Immunity|January 1, 2013
Interferon-inducible cholesterol-25-hydroxylase broadly inhibits viral entry by production of 25-hydroxycholesterolSu-Yang Liu, Roghiyh Aliyari, Kelechi Chikere, et al.
Nature Communications|January 10, 2017
Hepatitis C virus has a genetically determined lymphotropism through co-receptor B7.2Chia-Lin Chen, Jeffrey Y Huang, Chun-Hsiang Wang, et al.
The Journal of Biological Chemistry|October 6, 2005
The CATERPILLER protein monarch-1 is an antagonist of toll-like receptor-, tumor necrosis factor alpha-, and Mycobacterium tuberculosis-induced pro-inflammatory signalsKristi L Williams, John D Lich, Joseph A Duncan, et al.
Pageof 16