Showing results (1-10 of 184) with videos related to

Sort By:
Pageof 19
Biochimica Et Biophysica Acta|January 19, 2007
Cell cycle molecules define a pathway required for neuron death in development and diseaseLloyd A Greene, David X Liu, Carol M Troy, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 8, 2004
B-myb and C-myb play required roles in neuronal apoptosis evoked by nerve growth factor deprivation and DNA damageDavid X Liu, Subhas C Biswas, Lloyd A Greene
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 16, 2005
Bim is a direct target of a neuronal E2F-dependent apoptotic pathwaySubhas C Biswas, David X Liu, Lloyd A Greene
The Journal of Biological Chemistry|October 22, 2002
Nerve growth factor (NGF) down-regulates the Bcl-2 homology 3 (BH3) domain-only protein Bim and suppresses its proapoptotic activity by phosphorylationSubhas C Biswas, Lloyd A Greene
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|January 26, 2007
Bim is elevated in Alzheimer's disease neurons and is required for beta-amyloid-induced neuronal apoptosisSubhas C Biswas, Yijie Shi, Jean-Paul G Vonsattel, et al.
Neurochemical Research|March 21, 2008
Glucagon-like peptide-1 (GLP-1) diminishes neuronal degeneration and death caused by NGF deprivation by suppressing Bim inductionSubhas C Biswas, Jean Buteau, Lloyd A Greene
The Journal of Biological Chemistry|August 19, 2007
Pro-apoptotic Bim induction in response to nerve growth factor deprivation requires simultaneous activation of three different death signaling pathwaysSubhas C Biswas, Yijie Shi, Andrew Sproul, et al.
Neurochemical Research|September 28, 2005
Puma and p53 play required roles in death evoked in a cellular model of Parkinson diseaseSubhas C Biswas, Elizabeth Ryu, Clara Park, et al.
Cell Death Discovery|December 29, 2016
Role and regulation of Cdc25A phosphatase in neuron death induced by NGF deprivation or β-amyloidNandini Chatterjee, Priyankar Sanphui, Stav Kemeny, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 29, 2006
RTP801 is elevated in Parkinson brain substantia nigral neurons and mediates death in cellular models of Parkinson's disease by a mechanism involving mammalian target of rapamycin inactivationCristina Malagelada, Elizabeth J Ryu, Subhas C Biswas, et al.
Pageof 19