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Journal of Molecular and Cellular Cardiology|September 22, 2007
Sodium channel Scn1b null mice exhibit prolonged QT and RR intervalsLuis F Lopez-Santiago, Laurence S Meadows, Sara J Ernst, et al.
Redox Biology|October 2, 2022
Mitochondrial oxidative stress contributes to diastolic dysfunction through impaired mitochondrial dynamicsAndrey Lozhkin, Aleksandr E Vendrov, R Ramos-Mondragón, et al.
Circulation. Arrhythmia and Electrophysiology|December 10, 2016
Scn2b Deletion in Mice Results in Ventricular and Atrial ArrhythmiasYangyang Bao, B Cicero Willis, Chad R Frasier, et al.
JCI Insight|August 5, 2025
Altered Cardiac Excitability and Arrhythmia in Models of SCN1B-Linked Developmental and Epileptic EncephalopathyRoberto Ramos-Mondragon, Shuyun Wang, Nnamdi Edokobi, et al.
Brain : a Journal of Neurology|September 24, 2020
Variant-specific changes in persistent or resurgent sodium current in SCN8A-related epilepsy patient-derived neuronsAndrew M Tidball, Luis F Lopez-Santiago, Yukun Yuan, et al.
Elife|August 15, 2018
The adhesion function of the sodium channel beta subunit (β1) contributes to cardiac action potential propagationRengasayee Veeraraghavan, Gregory S Hoeker, Anita Alvarez-Laviada, et al.
Plos Biology|April 20, 2023
DSCAM gene triplication causes excessive GABAergic synapses in the neocortex in Down syndrome mouse modelsHao Liu, René N Caballero-Florán, Ty Hergenreder, et al.
JCI Insight|May 23, 2022
Neonatal Scn1b-null mice have sinoatrial node dysfunction, altered atrial structure, and atrial fibrillationRoberto Ramos-Mondragon, Nnamdi Edokobi, Samantha L Hodges, et al.
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