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Molecular and Cellular Biology|November 27, 2013
Mammary-specific gene activation is defined by progressive recruitment of STAT5 during pregnancy and the establishment of H3K4me3 marksKeunsoo Kang, Daisuke Yamaji, Kyung Hyun Yoo, et al.
The Journal of Experimental Medicine|April 1, 2009
Loss of STAT5 causes liver fibrosis and cancer development through increased TGF-{beta} and STAT3 activationAtsushi Hosui, Akiko Kimura, Daisuke Yamaji, et al.
Developmental Dynamics : an Official Publication of the American Association of Anatomists|January 6, 2007
Socs 3 modulates the activity of the transcription factor Stat3 in mammary tissue and controls alveolar homeostasisGertraud W Robinson, Margit Pacher-Zavisin, Bing Mei Zhu, et al.
Experimental & Molecular Medicine|April 24, 2020
The interdependence of mammary-specific super-enhancers and their native promoters facilitates gene activation during pregnancyXianke Zeng, Hye Kyung Lee, Chaochen Wang, et al.
International Journal of Molecular Sciences|February 13, 2026
STAT3R152W Mutation Model Reveals Temporal Changes in Hematopoietic PopulationsJakub Jankowski, Jichun Chen, Sung-Gwon Lee, et al.
Biology of Reproduction|January 30, 2004
A morphological and immunohistochemical comparison of mammary tissues from the short-tailed fruit bat (Carollia perspicillata) and the mouseJennifer L Evarts, John J Rasweiler, Richard R Behringer, et al.
Biochimica Et Biophysica Acta|June 30, 2007
The transcription factors Stat5a/b are not required for islet development but modulate pancreatic beta-cell physiology upon agingJi-Yeon Lee, Oksana Gavrilova, Behrous Davani, et al.
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