Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

Luís Relvas

Showing results (21-30 of 23) with videos related to

Pageof 3
Sort By:
You have reached the last page of results.This site can display upto 23 results.
Hemoglobin|February 2, 2013
Hb Plasencia [α125(H8)Leu→Arg (α2)] is a frequent cause of α+-thalassemia in the Portuguese populationElizabete Cunha, Celeste Bento, Ana Oliveira, et al.
Annals of Human Biology|January 15, 2011
SLC40A1 Q248H allele frequencies and associated SLC40A1 haplotypes in three West African population samplesDavid Albuquerque, Licínio Manco, Kovana M Loua, et al.
European Journal of Haematology|July 18, 2013
Molecular study of congenital erythrocytosis in 70 unrelated patients revealed a potential causal mutation in less than half of the cases (Where is/are the missing gene(s)?)Celeste Bento, Helena Almeida, Tabita M Maia, et al.
Pageof 3

Showing results (21-30 of 23) with videos related to

Sort By:
Pageof 3
You have reached the last page of results.This site can display upto 23 results.
Hemoglobin|February 2, 2013
Hb Plasencia [α125(H8)Leu→Arg (α2)] is a frequent cause of α+-thalassemia in the Portuguese populationElizabete Cunha, Celeste Bento, Ana Oliveira, et al.
Annals of Human Biology|January 15, 2011
SLC40A1 Q248H allele frequencies and associated SLC40A1 haplotypes in three West African population samplesDavid Albuquerque, Licínio Manco, Kovana M Loua, et al.
European Journal of Haematology|July 18, 2013
Molecular study of congenital erythrocytosis in 70 unrelated patients revealed a potential causal mutation in less than half of the cases (Where is/are the missing gene(s)?)Celeste Bento, Helena Almeida, Tabita M Maia, et al.
Pageof 3