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FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology
|
April 5, 2021
Slack K<sup>+</sup> channels attenuate NMDA-induced excitotoxic brain damage and neuronal cell death
Rebekka Ehinger, Anna Kuret, Lucas Matt, et al.
The Journal of Biological Chemistry
|
February 23, 2013
β2 adrenergic receptor, protein kinase A (PKA) and c-Jun N-terminal kinase (JNK) signaling pathways mediate tau pathology in Alzheimer disease models
Dayong Wang, Qin Fu, Yuan Zhou, et al.
Cellular and Molecular Life Sciences : CMLS
|
October 19, 2021
The Na<sup>+</sup>-activated K<sup>+</sup> channel Slack contributes to synaptic development and plasticity
Lucas Matt, Thomas Pham, David Skrabak, et al.
JCI Insight
|
March 17, 2026
Slick K+ channels contribute to cardiac remodeling, fibrosis and dysfunction in post-infarction hearts
Jiaqi Yang, Lin Zhu, David Spähn, et al.
Eneuro
|
November 2, 2016
Loss of SynDIG1 Reduces Excitatory Synapse Maturation But Not Formation <i>In Vivo</i>
George Chenaux, Lucas Matt, Travis C Hill, et al.
Iscience
|
April 19, 2021
Potassium ions promote hexokinase-II dependent glycolysis
Helmut Bischof, Sandra Burgstaller, Anna Springer, et al.
Iscience
|
May 20, 2021
Erratum: Potassium ions promote hexokinase-II dependent glycolysis
Helmut Bischof, Sandra Burgstaller, Anna Springer, et al.
Cellular and Molecular Life Sciences : CMLS
|
November 21, 2023
BK channels sustain neuronal Ca<sup>2+</sup> oscillations to support hippocampal long-term potentiation and memory formation
Thomas Pham, Tamara Hussein, Dila Calis, et al.
Science Signaling
|
January 26, 2017
Phosphorylation of Ser1928 mediates the enhanced activity of the L-type Ca2+ channel Cav1.2 by the β2-adrenergic receptor in neurons
Hai Qian, Tommaso Patriarchi, Jennifer L Price, et al.
Cell Reports
|
March 1, 2018
SynDIG4/Prrt1 Is Required for Excitatory Synapse Development and Plasticity Underlying Cognitive Function
Lucas Matt, Lyndsey M Kirk, George Chenaux, et al.
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Search research articles
Search
Showing results (11-20 of 27) with videos related to
Sort By:
Page
of 3
FASEB Journal : Official Publication of the Federation of American Societies for Experimental Biology
|
April 5, 2021
Slack K<sup>+</sup> channels attenuate NMDA-induced excitotoxic brain damage and neuronal cell death
Rebekka Ehinger, Anna Kuret, Lucas Matt, et al.
The Journal of Biological Chemistry
|
February 23, 2013
β2 adrenergic receptor, protein kinase A (PKA) and c-Jun N-terminal kinase (JNK) signaling pathways mediate tau pathology in Alzheimer disease models
Dayong Wang, Qin Fu, Yuan Zhou, et al.
Cellular and Molecular Life Sciences : CMLS
|
October 19, 2021
The Na<sup>+</sup>-activated K<sup>+</sup> channel Slack contributes to synaptic development and plasticity
Lucas Matt, Thomas Pham, David Skrabak, et al.
JCI Insight
|
March 17, 2026
Slick K+ channels contribute to cardiac remodeling, fibrosis and dysfunction in post-infarction hearts
Jiaqi Yang, Lin Zhu, David Spähn, et al.
Eneuro
|
November 2, 2016
Loss of SynDIG1 Reduces Excitatory Synapse Maturation But Not Formation <i>In Vivo</i>
George Chenaux, Lucas Matt, Travis C Hill, et al.
Iscience
|
April 19, 2021
Potassium ions promote hexokinase-II dependent glycolysis
Helmut Bischof, Sandra Burgstaller, Anna Springer, et al.
Iscience
|
May 20, 2021
Erratum: Potassium ions promote hexokinase-II dependent glycolysis
Helmut Bischof, Sandra Burgstaller, Anna Springer, et al.
Cellular and Molecular Life Sciences : CMLS
|
November 21, 2023
BK channels sustain neuronal Ca<sup>2+</sup> oscillations to support hippocampal long-term potentiation and memory formation
Thomas Pham, Tamara Hussein, Dila Calis, et al.
Science Signaling
|
January 26, 2017
Phosphorylation of Ser1928 mediates the enhanced activity of the L-type Ca2+ channel Cav1.2 by the β2-adrenergic receptor in neurons
Hai Qian, Tommaso Patriarchi, Jennifer L Price, et al.
Cell Reports
|
March 1, 2018
SynDIG4/Prrt1 Is Required for Excitatory Synapse Development and Plasticity Underlying Cognitive Function
Lucas Matt, Lyndsey M Kirk, George Chenaux, et al.
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of 3