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The Journal of Physiology|December 3, 2015
Contribution of postsynaptic T-type calcium channels to parallel fibre-Purkinje cell synaptic responsesRomain Ly, Guy Bouvier, German Szapiro, et al.
Neuron|September 16, 2014
Activity-dependent gating of calcium spikes by A-type K+ channels controls climbing fiber signaling in Purkinje cell dendritesYo Otsu, Païkan Marcaggi, Anne Feltz, et al.
Cellular Microbiology|October 8, 2014
Epsilon toxin from Clostridium perfringens acts on oligodendrocytes without forming pores, and causes demyelinationLaetitia Wioland, Jean-Luc Dupont, Frédéric Doussau, et al.
Elife|February 6, 2020
Gradients in the mammalian cerebellar cortex enable Fourier-like transformation and improve storing capacityIsabelle Straub, Laurens Witter, Abdelmoneim Eshra, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 7, 2009
Functional coupling between mGluR1 and Cav3.1 T-type calcium channels contributes to parallel fiber-induced fast calcium signaling within Purkinje cell dendritic spinesMichael E Hildebrand, Philippe Isope, Taisuke Miyazaki, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 19, 2013
Clusters of cerebellar Purkinje cells control their afferent climbing fiber dischargeJoseph Chaumont, Nicolas Guyon, Antoine M Valera, et al.
Elife|March 4, 2021
Sushi domain-containing protein 4 controls synaptic plasticity and motor learningInés González-Calvo, Keerthana Iyer, Mélanie Carquin, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|April 23, 2021
SCA7 Mouse Cerebellar Pathology Reveals Preferential Downregulation of Key Purkinje Cell-Identity Genes and Shared Disease Signature with SCA1 and SCA2Anna Niewiadomska-Cimicka, Frédéric Doussau, Jean-Baptiste Perot, et al.
Neuron|May 15, 2012
Relevance of exocytotic glutamate release from retinal gliaMichal Slezak, Antje Grosche, Aurore Niemiec, et al.
Molecular Cell|August 9, 2016
Cerebellar Ataxia and Coenzyme Q Deficiency through Loss of Unorthodox Kinase ActivityJonathan A Stefely, Floriana Licitra, Leila Laredj, et al.
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