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Research Square|February 23, 2026
Type-2-immune history imprints local training in nerve-airway associated interstitial macrophages (NAMs) for disease tolerance during respiratory viral infectionPayal Damani-Yokota, Yavor Yordanov, Eduardo D Bernier, et al.Circulation|March 5, 2008
Deletion of Ptpn11 (Shp2) in cardiomyocytes causes dilated cardiomyopathy via effects on the extracellular signal-regulated kinase/mitogen-activated protein kinase and RhoA signaling pathwaysMaria I Kontaridis, Wentian Yang, Kendra K Bence, et al.Cancer Cell|September 14, 2010
The Notch/Hes1 pathway sustains NF-κB activation through CYLD repression in T cell leukemiaLluis Espinosa, Severine Cathelin, Teresa D'Altri, et al.Cell Systems|April 15, 2019
N-Glycoproteomics of Patient-Derived Xenografts: A Strategy to Discover Tumor-Associated Proteins in High-Grade Serous Ovarian CancerAnkit Sinha, Ali Hussain, Vladimir Ignatchenko, et al.The Journal of Biological Chemistry|September 16, 2010
Altered glucose homeostasis in mice with liver-specific deletion of Src homology phosphatase 2Kosuke Matsuo, Mirela Delibegovic, Izumi Matsuo, et al.Plos One|June 2, 2012
Regulation of signaling at regions of cell-cell contact by endoplasmic reticulum-bound protein-tyrosine phosphatase 1BFawaz G Haj, Ola Sabet, Ali Kinkhabwala, et al.Human Molecular Genetics|April 17, 2018
Noonan syndrome-causing SHP2 mutants impair ERK-dependent chondrocyte differentiation during endochondral bone growthMylène Tajan, Julie Pernin-Grandjean, Nicolas Beton, et al.Leukemia|November 28, 2019
Therapeutic targeting of the E3 ubiquitin ligase SKP2 in T-ALLSonia Rodriguez, Christina Abundis, Francesco Boccalatte, et al.Pharmacology & Therapeutics|August 3, 2019
Machine learning and data mining frameworks for predicting drug response in cancer: An overview and a novel in silico screening process based on association rule miningKonstantinos Vougas, Theodore Sakellaropoulos, Athanassios Kotsinas, et al.Proceedings of the National Academy of Sciences of the United States of America|May 6, 2015
Oncogenic and RASopathy-associated K-RAS mutations relieve membrane-dependent occlusion of the effector-binding siteMohammad T Mazhab-Jafari, Christopher B Marshall, Matthew J Smith, et al.Pageof 43