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Chemistry & Biology|March 29, 2011
JAK3 specific kinase inhibitors: when specificity is not enoughLuk Cox, Jan CoolsRNA (New York, N.Y.)|April 28, 2012
Development of a siRNA and shRNA screening system based on a kinase fusion proteinSofie Pieraets, Luk Cox, Olga Gielen, et al.International Journal of Cardiology|August 22, 2008
A broken heart: a stretch too far: an overview of mouse models with mutations in stretch-sensor componentsLuk Cox, Lieve Umans, Frederique Cornelis, et al.The American Journal of Pathology|April 26, 2007
Inactivation of Smad5 in endothelial cells and smooth muscle cells demonstrates that Smad5 is required for cardiac homeostasisLieve Umans, Luk Cox, Marc Tjwa, et al.Haematologica|July 23, 2013
NUP214-ABL1-mediated cell proliferation in T-cell acute lymphoblastic leukemia is dependent on the LCK kinase and various interacting proteinsKim De Keersmaecker, Michaël Porcu, Luk Cox, et al.Molecular Oncology|March 13, 2014
The tyrosine phosphatase SHP2 is required for cell transformation by the receptor tyrosine kinase mutants FIP1L1-PDGFRα and PDGFRα D842VLaura A Noël, Florence A Arts, Carmen P Montano-Almendras, et al.Blood|February 18, 2011
JAK2 rearrangements, including the novel SEC31A-JAK2 fusion, are recurrent in classical Hodgkin lymphomaKatrien Van Roosbroeck, Luk Cox, Thomas Tousseyn, et al.Blood|September 7, 2014
JAK3 mutants transform hematopoietic cells through JAK1 activation, causing T-cell acute lymphoblastic leukemia in a mouse modelSandrine Degryse, Charles E de Bock, Luk Cox, et al.Neurobiology of Disease|August 1, 2006
Ozzy, a Jag1 vestibular mouse mutant, displays characteristics of Alagille syndromeKaren Vrijens, Sofie Thys, Marcel T De Jeu, et al.Circulation Research|July 23, 2005
Alk3/Bmpr1a receptor is required for development of the atrioventricular canal into valves and annulus fibrosusVinciane Gaussin, Gregory E Morley, Luk Cox, et al.Pageof 2