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Luke C Davies

Showing results (11-20 of 33) with videos related to

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European Journal of Immunology|January 27, 2011
Hoxb8 conditionally immortalised macrophage lines model inflammatory monocytic cells with important similarity to dendritic cellsMarcela Rosas, Fabiola Osorio, Matthew J Robinson, et al.
Journal of Visualized Experiments : Jove|March 4, 2024
Lentiviral Vector Preparation for Efficient Gene and MicroRNA Modulation of Peritoneal Cavity Tissue-Resident Macrophages In Vivo in MiceMark Gurney, Luke C Davies, Ruth E Jones, et al.
Redox Biology|September 28, 2016
Autocrine IL-10 functions as a rheostat for M1 macrophage glycolytic commitment by tuning nitric oxide productionWalter A Baseler, Luke C Davies, Laura Quigley, et al.
The American Journal of Pathology|February 11, 2014
miR-192 induces G2/M growth arrest in aristolochic acid nephropathyRobert H Jenkins, Luke C Davies, Philip R Taylor, et al.
The Journal of Clinical Investigation|June 20, 2018
Itaconic acid mediates crosstalk between macrophage metabolism and peritoneal tumorsJonathan M Weiss, Luke C Davies, Megan Karwan, et al.
Nature Communications|May 23, 2013
Distinct bone marrow-derived and tissue-resident macrophage lineages proliferate at key stages during inflammationLuke C Davies, Marcela Rosas, Stephen J Jenkins, et al.
Science (New York, N.Y.)|April 26, 2014
The transcription factor Gata6 links tissue macrophage phenotype and proliferative renewalMarcela Rosas, Luke C Davies, Peter J Giles, et al.
European Journal of Immunology|July 6, 2016
IL-10 differentially controls the infiltration of inflammatory macrophages and antigen-presenting cells during inflammationChia-Te Liao, Marcela Rosas, Luke C Davies, et al.
The EMBO Journal|June 3, 2020
Tissue-resident macrophages actively suppress IL-1beta release via a reactive prostanoid/IL-10 pathwayNatacha Ipseiz, Robert J Pickering, Marcela Rosas, et al.
Plos Pathogens|June 27, 2019
The protective effect of inflammatory monocytes during systemic C. albicans infection is dependent on collaboration between C-type lectin-like receptorsAiysha Thompson, Luke C Davies, Chia-Te Liao, et al.
Pageof 4

Showing results (11-20 of 33) with videos related to

Sort By:
Pageof 4
European Journal of Immunology|January 27, 2011
Hoxb8 conditionally immortalised macrophage lines model inflammatory monocytic cells with important similarity to dendritic cellsMarcela Rosas, Fabiola Osorio, Matthew J Robinson, et al.
Journal of Visualized Experiments : Jove|March 4, 2024
Lentiviral Vector Preparation for Efficient Gene and MicroRNA Modulation of Peritoneal Cavity Tissue-Resident Macrophages In Vivo in MiceMark Gurney, Luke C Davies, Ruth E Jones, et al.
Redox Biology|September 28, 2016
Autocrine IL-10 functions as a rheostat for M1 macrophage glycolytic commitment by tuning nitric oxide productionWalter A Baseler, Luke C Davies, Laura Quigley, et al.
The American Journal of Pathology|February 11, 2014
miR-192 induces G2/M growth arrest in aristolochic acid nephropathyRobert H Jenkins, Luke C Davies, Philip R Taylor, et al.
The Journal of Clinical Investigation|June 20, 2018
Itaconic acid mediates crosstalk between macrophage metabolism and peritoneal tumorsJonathan M Weiss, Luke C Davies, Megan Karwan, et al.
Nature Communications|May 23, 2013
Distinct bone marrow-derived and tissue-resident macrophage lineages proliferate at key stages during inflammationLuke C Davies, Marcela Rosas, Stephen J Jenkins, et al.
Science (New York, N.Y.)|April 26, 2014
The transcription factor Gata6 links tissue macrophage phenotype and proliferative renewalMarcela Rosas, Luke C Davies, Peter J Giles, et al.
European Journal of Immunology|July 6, 2016
IL-10 differentially controls the infiltration of inflammatory macrophages and antigen-presenting cells during inflammationChia-Te Liao, Marcela Rosas, Luke C Davies, et al.
The EMBO Journal|June 3, 2020
Tissue-resident macrophages actively suppress IL-1beta release via a reactive prostanoid/IL-10 pathwayNatacha Ipseiz, Robert J Pickering, Marcela Rosas, et al.
Plos Pathogens|June 27, 2019
The protective effect of inflammatory monocytes during systemic C. albicans infection is dependent on collaboration between C-type lectin-like receptorsAiysha Thompson, Luke C Davies, Chia-Te Liao, et al.
Pageof 4