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Lynn Bedford

Showing results (1-10 of 20) with videos related to

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Essays in Biochemistry|October 28, 2005
The ubiquitin-proteasome system and neurodegenerative disordersRobert Layfield, James Lowe, Lynn Bedford
Biochemical Society Transactions|July 27, 2011
Can neurodegeneration be separated from neuropathological hallmarks of chronic idiopathic human neurodegenerative disease? A perspective from modelling!Simon Paine, James Lowe, Lynn Bedford, et al.
Neuroscience Letters|January 11, 2011
Diverse polyubiquitin chains accumulate following 26S proteasomal dysfunction in mammalian neuronesLynn Bedford, Robert Layfield, R John Mayer, et al.
Trends in Cell Biology|April 30, 2010
Assembly, structure, and function of the 26S proteasomeLynn Bedford, Simon Paine, Paul W Sheppard, et al.
Nature Reviews. Drug Discovery|December 15, 2010
Ubiquitin-like protein conjugation and the ubiquitin-proteasome system as drug targetsLynn Bedford, James Lowe, Lawrence R Dick, et al.
Neuroscience Letters|June 9, 2012
Heterozygosity for the proteasomal Psmc1 ATPase is insufficient to cause neuropathology in mouse brain, but causes cell cycle defects in mouse embryonic fibroblastsNooshin Rezvani, Jamal Elkharaz, Karen Lawler, et al.
Autophagy|December 17, 2008
The UPS and autophagy in chronic neurodegenerative disease: six of one and half a dozen of the other--or not?Lynn Bedford, Simon Paine, Nooshin Rezvani, et al.
Developmental Biology|May 11, 2005
Id4 is required for the correct timing of neural differentiationLynn Bedford, Robert Walker, Toru Kondo, et al.
Biochimica Et Biophysica Acta|November 4, 2008
Is malfunction of the ubiquitin proteasome system the primary cause of alpha-synucleinopathies and other chronic human neurodegenerative disease?Lynn Bedford, David Hay, Simon Paine, et al.
Plos One|February 6, 2013
Pale body-like inclusion formation and neurodegeneration following depletion of 26S proteasomes in mouse brain neurones are independent of α-synucleinSimon M L Paine, Glenn Anderson, Karen Bedford, et al.
Pageof 2

Showing results (1-10 of 20) with videos related to

Sort By:
Pageof 2
Essays in Biochemistry|October 28, 2005
The ubiquitin-proteasome system and neurodegenerative disordersRobert Layfield, James Lowe, Lynn Bedford
Biochemical Society Transactions|July 27, 2011
Can neurodegeneration be separated from neuropathological hallmarks of chronic idiopathic human neurodegenerative disease? A perspective from modelling!Simon Paine, James Lowe, Lynn Bedford, et al.
Neuroscience Letters|January 11, 2011
Diverse polyubiquitin chains accumulate following 26S proteasomal dysfunction in mammalian neuronesLynn Bedford, Robert Layfield, R John Mayer, et al.
Trends in Cell Biology|April 30, 2010
Assembly, structure, and function of the 26S proteasomeLynn Bedford, Simon Paine, Paul W Sheppard, et al.
Nature Reviews. Drug Discovery|December 15, 2010
Ubiquitin-like protein conjugation and the ubiquitin-proteasome system as drug targetsLynn Bedford, James Lowe, Lawrence R Dick, et al.
Neuroscience Letters|June 9, 2012
Heterozygosity for the proteasomal Psmc1 ATPase is insufficient to cause neuropathology in mouse brain, but causes cell cycle defects in mouse embryonic fibroblastsNooshin Rezvani, Jamal Elkharaz, Karen Lawler, et al.
Autophagy|December 17, 2008
The UPS and autophagy in chronic neurodegenerative disease: six of one and half a dozen of the other--or not?Lynn Bedford, Simon Paine, Nooshin Rezvani, et al.
Developmental Biology|May 11, 2005
Id4 is required for the correct timing of neural differentiationLynn Bedford, Robert Walker, Toru Kondo, et al.
Biochimica Et Biophysica Acta|November 4, 2008
Is malfunction of the ubiquitin proteasome system the primary cause of alpha-synucleinopathies and other chronic human neurodegenerative disease?Lynn Bedford, David Hay, Simon Paine, et al.
Plos One|February 6, 2013
Pale body-like inclusion formation and neurodegeneration following depletion of 26S proteasomes in mouse brain neurones are independent of α-synucleinSimon M L Paine, Glenn Anderson, Karen Bedford, et al.
Pageof 2