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Nature Communications|September 6, 2017
Editing an α-globin enhancer in primary human hematopoietic stem cells as a treatment for β-thalassemiaSachith Mettananda, Chris A Fisher, Deborah Hay, et al.Science Advances|April 14, 2023
M2 macrophages drive leukemic transformation by imposing resistance to phagocytosis and improving mitochondrial metabolismIsabel Weinhäuser, Diego A Pereira-Martins, Luciana Y Almeida, et al.Cell Reports. Medicine|January 9, 2026
ΔNp73 isoform defines a TP53-mutant-like poor-risk subgroup of acute myeloid leukemiaDiego A Pereira-Martins, Cesar Ortiz, Isabel Weinhäuser, et al.Cell Reports|May 12, 2021
Chromatin accessibility governs the differential response of cancer and T cells to arginine starvationNicholas T Crump, Andreas V Hadjinicolaou, Meng Xia, et al.Nature Medicine|July 18, 2018
Clonal heterogeneity of acute myeloid leukemia treated with the IDH2 inhibitor enasidenibLynn Quek, Muriel D David, Alison Kennedy, et al.American Journal of Hematology|November 27, 2023
Sickle cell allele HBB-rs334(T) is associated with decreased risk of childhood Burkitt lymphoma in East AfricaHyokyoung G Hong, Mateus H Gouveia, Martin D Ogwang, et al.Nature Genetics|March 23, 2011
Systematic documentation and analysis of human genetic variation in hemoglobinopathies using the microattribution approachBelinda Giardine, Joseph Borg, Douglas R Higgs, et al.Cancer Cell|January 22, 2011
Coexistence of LMPP-like and GMP-like leukemia stem cells in acute myeloid leukemiaNicolas Goardon, Emanuele Marchi, Ann Atzberger, et al.The Journal of Experimental Medicine|July 6, 2016
Genetically distinct leukemic stem cells in human CD34- acute myeloid leukemia are arrested at a hemopoietic precursor-like stageLynn Quek, Georg W Otto, Catherine Garnett, et al.Blood Advances|May 8, 2020
Molecular mechanisms mediating relapse following ivosidenib monotherapy in IDH1-mutant relapsed or refractory AMLSung Choe, Hongfang Wang, Courtney D DiNardo, et al.Pageof 20