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Nature Medicine|July 18, 2018
Clonal heterogeneity of acute myeloid leukemia treated with the IDH2 inhibitor enasidenibLynn Quek, Muriel D David, Alison Kennedy, et al.Cancer Cell|January 22, 2011
Coexistence of LMPP-like and GMP-like leukemia stem cells in acute myeloid leukemiaNicolas Goardon, Emanuele Marchi, Ann Atzberger, et al.The Journal of Experimental Medicine|July 6, 2016
Genetically distinct leukemic stem cells in human CD34- acute myeloid leukemia are arrested at a hemopoietic precursor-like stageLynn Quek, Georg W Otto, Catherine Garnett, et al.Blood Advances|May 8, 2020
Molecular mechanisms mediating relapse following ivosidenib monotherapy in IDH1-mutant relapsed or refractory AMLSung Choe, Hongfang Wang, Courtney D DiNardo, et al.Signal Transduction and Targeted Therapy|July 13, 2025
High mtDNA content identifies oxidative phosphorylation-driven acute myeloid leukemias and represents a therapeutic vulnerabilityDiego A Pereira-Martins, Isabel Weinhäuser, Emmanuel Griessinger, et al.Blood|September 14, 2013
Clinical and biological implications of driver mutations in myelodysplastic syndromesElli Papaemmanuil, Moritz Gerstung, Luca Malcovati, et al.Leukemia|January 13, 2019
TP53 mutation status divides myelodysplastic syndromes with complex karyotypes into distinct prognostic subgroupsDetlef Haase, Kristen E Stevenson, Donna Neuberg, et al.Nature Genetics|May 19, 2015
Factors influencing success of clinical genome sequencing across a broad spectrum of disordersJenny C Taylor, Hilary C Martin, Stefano Lise, et al.Pageof 3