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Magyar Onkologia|September 21, 2017
[New recommendations in oncocardiology]Csaba András Nagy, Mária Lódi, Ingrid Balogh, et al.
Orvosi Hetilap|November 15, 2017
[Ischaemic heart disease in cancer patients. Questions and problems]Petra Gulácsi-Bárdos, Miklós Szokol, Mária Lódi, et al.
Frontiers in Physiology|May 1, 2020
Modulation of Titin-Based Stiffness in Hypertrophic Cardiomyopathy via Protein Kinase DMelissa Herwig, Detmar Kolijn, Mária Lódi, et al.
Journal of Translational Medicine|December 10, 2020
Prophylactic, single-drug cardioprotection in a comparative, experimental study of doxorubicin-induced cardiomyopathyMária Lódi, Viktor Bánhegyi, Beáta Bódi, et al.
Frontiers in Physiology|June 12, 2020
Enhanced Cardiomyocyte Function in Hypertensive Rats With Diastolic Dysfunction and Human Heart Failure Patients After Acute Treatment With Soluble Guanylyl Cyclase (sGC) ActivatorDetmar Kolijn, Árpád Kovács, Melissa Herwig, et al.
Journal of Translational Medicine|July 21, 2019
Advantages of prophylactic versus conventionally scheduled heart failure therapy in an experimental model of doxorubicin-induced cardiomyopathyMária Lódi, Dániel Priksz, Gábor Áron Fülöp, et al.
Cells|August 7, 2021
Human Tissue Angiotensin Converting Enzyme (ACE) Activity Is Regulated by Genetic Polymorphisms, Posttranslational Modifications, Endogenous Inhibitors and Secretion in the Serum, Lungs and HeartViktor Bánhegyi, Attila Enyedi, Gábor Áron Fülöp, et al.
Antioxidants (Basel, Switzerland)|August 6, 2021
The Interplay between S-Glutathionylation and Phosphorylation of Cardiac Troponin I and Myosin Binding Protein C in End-Stage Human Failing HeartsHeidi Budde, Roua Hassoun, Melina Tangos, et al.
International Journal of Cardiology|September 13, 2021
Stress activated signalling impaired protein quality control pathways in human hypertrophic cardiomyopathyRoua Hassoun, Heidi Budde, Saltanat Zhazykbayeva, et al.
International Journal of Molecular Sciences|September 10, 2021
De Novo Missense Mutations in <i>TNNC1</i> and <i>TNNI3</i> Causing Severe Infantile Cardiomyopathy Affect Myofilament Structure and Function and Are Modulated by Troponin Targeting AgentsRoua Hassoun, Heidi Budde, Hans Georg Mannherz, et al.
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