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M Argast

Showing results (11-20 of 24) with videos related to

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Journal of Molecular Biology|July 17, 1998
I-PpoI and I-CreI homing site sequence degeneracy determined by random mutagenesis and sequential in vitro enrichmentG M Argast, K M Stephens, M J Emond, et al.
The International Journal of Biochemistry & Cell Biology|August 17, 2010
Inactivation of p38 MAPK during liver regenerationJean S Campbell, Gretchen M Argast, Sebastian Y Yuen, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 17, 1996
Expression and characterization of glycogen synthase kinase-3 mutants and their effect on glycogen synthase activity in intact cellsH Eldar-Finkelman, G M Argast, O Foord, et al.
Science (New York, N.Y.)|April 19, 2008
Wnt5a control of cell polarity and directional movement by polarized redistribution of adhesion receptorsEric S Witze, Elizabeth S Litman, Gretchen M Argast, et al.
Oncogene|June 2, 2009
Plexin B1 is repressed by oncogenic B-Raf signaling and functions as a tumor suppressor in melanoma cellsG M Argast, C H Croy, K L Couts, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 1, 1995
cAMP- and rapamycin-sensitive regulation of the association of eukaryotic initiation factor 4E and the translational regulator PHAS-I in aortic smooth muscle cellsL M Graves, K E Bornfeldt, G M Argast, et al.
Anticancer Research|January 31, 2012
The evaluation of E-Cadherin and vimentin as biomarkers of clinical outcomes among patients with non-small cell lung cancer treated with erlotinib as second- or third-line therapyFrank Richardson, G David Young, Regina Sennello, et al.
The Journal of Clinical Investigation|August 15, 1997
The mitogen-activated protein kinase pathway can mediate growth inhibition and proliferation in smooth muscle cells. Dependence on the availability of downstream targetsK E Bornfeldt, J S Campbell, H Koyama, et al.
The Journal of Biological Chemistry|January 5, 1996
Platelet-derived growth factor stimulates protein kinase A through a mitogen-activated protein kinase-dependent pathway in human arterial smooth muscle cellsL M Graves, K E Bornfeldt, J S Sidhu, et al.
The Journal of Clinical Investigation|May 26, 2001
Expression of suppressors of cytokine signaling during liver regenerationJ S Campbell, L Prichard, F Schaper, et al.
Pageof 3

Showing results (11-20 of 24) with videos related to

Sort By:
Pageof 3
Journal of Molecular Biology|July 17, 1998
I-PpoI and I-CreI homing site sequence degeneracy determined by random mutagenesis and sequential in vitro enrichmentG M Argast, K M Stephens, M J Emond, et al.
The International Journal of Biochemistry & Cell Biology|August 17, 2010
Inactivation of p38 MAPK during liver regenerationJean S Campbell, Gretchen M Argast, Sebastian Y Yuen, et al.
Proceedings of the National Academy of Sciences of the United States of America|September 17, 1996
Expression and characterization of glycogen synthase kinase-3 mutants and their effect on glycogen synthase activity in intact cellsH Eldar-Finkelman, G M Argast, O Foord, et al.
Science (New York, N.Y.)|April 19, 2008
Wnt5a control of cell polarity and directional movement by polarized redistribution of adhesion receptorsEric S Witze, Elizabeth S Litman, Gretchen M Argast, et al.
Oncogene|June 2, 2009
Plexin B1 is repressed by oncogenic B-Raf signaling and functions as a tumor suppressor in melanoma cellsG M Argast, C H Croy, K L Couts, et al.
Proceedings of the National Academy of Sciences of the United States of America|August 1, 1995
cAMP- and rapamycin-sensitive regulation of the association of eukaryotic initiation factor 4E and the translational regulator PHAS-I in aortic smooth muscle cellsL M Graves, K E Bornfeldt, G M Argast, et al.
Anticancer Research|January 31, 2012
The evaluation of E-Cadherin and vimentin as biomarkers of clinical outcomes among patients with non-small cell lung cancer treated with erlotinib as second- or third-line therapyFrank Richardson, G David Young, Regina Sennello, et al.
The Journal of Clinical Investigation|August 15, 1997
The mitogen-activated protein kinase pathway can mediate growth inhibition and proliferation in smooth muscle cells. Dependence on the availability of downstream targetsK E Bornfeldt, J S Campbell, H Koyama, et al.
The Journal of Biological Chemistry|January 5, 1996
Platelet-derived growth factor stimulates protein kinase A through a mitogen-activated protein kinase-dependent pathway in human arterial smooth muscle cellsL M Graves, K E Bornfeldt, J S Sidhu, et al.
The Journal of Clinical Investigation|May 26, 2001
Expression of suppressors of cytokine signaling during liver regenerationJ S Campbell, L Prichard, F Schaper, et al.
Pageof 3