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Neuropathology and Applied Neurobiology|June 1, 1994
Herpes simplex virus type 1 gene expression and reactivation of latent infection in the central nervous systemI Steiner, N Mador, I Reibstein, et al.Journal of Virology|September 1, 1988
Latent herpes simplex virus type 1 transcription in human trigeminal gangliaI Steiner, J G Spivack, D R O'Boyle, et al.Journal of Virology|March 1, 1988
Latent herpes simplex virus type 1 transcripts in peripheral and central nervous system tissues of mice map to similar regions of the viral genomeA M Deatly, J G Spivack, E Lavi, et al.Virus Research|July 1, 1991
The HSV-1 latency associated transcript (LAT) variants 1704 and 1705 are glycoprotein C negativeZ Wroblewska, J G Spivack, J Otte, et al.Journal of Virology|April 1, 1990
A herpes simplex virus type 1 mutant containing a nontransinducing Vmw65 protein establishes latent infection in vivo in the absence of viral replication and reactivates efficiently from explanted trigeminal gangliaI Steiner, J G Spivack, S L Deshmane, et al.The American Psychologist|October 1, 1993
The science of prevention. A conceptual framework and some directions for a national research programJ D Coie, N F Watt, S G West, et al.Journal of Virology|July 1, 1990
A herpes simplex virus type 1 latency-associated transcript mutant reactivates with normal kinetics from latent infectionT M Block, J G Spivack, I Steiner, et al.The Journal of General Virology|March 1, 1991
Investigation of herpes simplex virus type 1 (HSV-1) gene expression and DNA synthesis during the establishment of latent infection by an HSV-1 mutant, in1814, that does not replicate in mouse trigeminal gangliaT Valyi-Nagy, S L Deshmane, J G Spivack, et al.Current Eye Research|January 1, 1991
A review of the molecular mechanism of HSV-1 latencyN W Fraser, J G Spivack, Z Wroblewska, et al.The EMBO Journal|February 1, 1989
Herpes simplex virus type 1 latency-associated transcripts are evidently not essential for latent infectionI Steiner, J G Spivack, R P Lirette, et al.Pageof 5