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Proceedings of the National Academy of Sciences of the United States of America|November 11, 1992
IRS-1 activates phosphatidylinositol 3'-kinase by associating with src homology 2 domains of p85M G Myers, J M Backer, X J Sun, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|June 23, 2012
Molecular imaging of vascular endothelial growth factor receptors in graft arteriosclerosisJiasheng Zhang, Mahmoud Razavian, Sina Tavakoli, et al.
The Journal of Cell Biology|September 1, 2004
Phospholipase C and cofilin are required for carcinoma cell directionality in response to EGF stimulationGhassan Mouneimne, Lilian Soon, Vera DesMarais, et al.
Cancer Research|March 26, 2016
GPCR Signaling Mediates Tumor Metastasis via PI3KβBassem D Khalil, Christine Hsueh, Yanyan Cao, et al.
Journal of Controlled Release : Official Journal of the Controlled Release Society|May 10, 2003
Humanized docking system for assembly of targeting drug delivery complexesMarina V Backer, Timur I Gaynutdinov, Inna I Gorshkova, et al.
Science Signaling|August 18, 2016
Coincident signals from GPCRs and receptor tyrosine kinases are uniquely transduced by PI3Kβ in myeloid cellsDaniel M Houslay, Karen E Anderson, Tamara Chessa, et al.
Proceedings of the National Academy of Sciences of the United States of America|April 1, 2015
Phosphatidylinositol 4-phosphate and phosphatidylinositol 3-phosphate regulate phagolysosome biogenesisAndreas Jeschke, Nicole Zehethofer, Buko Lindner, et al.
The Journal of Cell Biology|August 1, 1992
The insulin receptor juxtamembrane region contains two independent tyrosine/beta-turn internalization signalsJ M Backer, S E Shoelson, M A Weiss, et al.
Experimental Cell Research|March 5, 2008
RhoA/ROCK-mediated switching between Cdc42- and Rac1-dependent protrusion in MTLn3 carcinoma cellsMirvat El-Sibai, Olivier Pertz, Huan Pang, et al.
Scientific Reports|January 27, 2021
Absence of S100A4 in the mouse lens induces an aberrant retina-specific differentiation program and cataractRupalatha Maddala, Junyuan Gao, Richard T Mathias, et al.
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