Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Filters

M Baes

Showing results (31-40 of 38) with videos related to

Pageof 4
Sort By:
You have reached the last page of results.This site can display upto 38 results.
Annals of Neurology|September 8, 2000
Neuronal migration disorder in Zellweger mice is secondary to glutamate receptor dysfunctionP Gressens, M Baes, P Leroux, et al.
The Biochemical Journal|February 15, 2001
Prenatal and postnatal development of peroxisomal lipid-metabolizing pathways in the mouseS Huyghe, M Casteels, A Janssen, et al.
Nature Genetics|December 17, 1997
Urokinase-generated plasmin activates matrix metalloproteinases during aneurysm formationP Carmeliet, L Moons, R Lijnen, et al.
European Journal of Biochemistry|July 15, 1996
Rat pristanoyl-CoA oxidase. cDNA cloning and recognition of its C-terminal (SQL) by the peroxisomal-targeting signal 1 receptorJ C Vanhooren, M Fransen, B de Béthune, et al.
Nature Genetics|September 1, 1997
A mouse model for Zellweger syndromeM Baes, P Gressens, E Baumgart, et al.
Journal of Neuropathology and Experimental Neurology|April 10, 2002
The neuronal migration defect in mice with Zellweger syndrome (Pex5 knockout) is not caused by the inactivity of peroxisomal beta-oxidationM Baes, P Gressens, S Huyghe, et al.
Cellular and Molecular Life Sciences : CMLS|June 23, 2006
Farnesylation of Pex19p is not essential for peroxisome biogenesis in yeast and mammalian cellsI M K Vastiau, E A Anthonio, M Brams, et al.
Nature Medicine|September 30, 1999
Inhibition of plasminogen activators or matrix metalloproteinases prevents cardiac rupture but impairs therapeutic angiogenesis and causes cardiac failureS Heymans, A Luttun, D Nuyens, et al.
Pageof 4

Showing results (31-40 of 38) with videos related to

Sort By:
Pageof 4
You have reached the last page of results.This site can display upto 38 results.
Annals of Neurology|September 8, 2000
Neuronal migration disorder in Zellweger mice is secondary to glutamate receptor dysfunctionP Gressens, M Baes, P Leroux, et al.
The Biochemical Journal|February 15, 2001
Prenatal and postnatal development of peroxisomal lipid-metabolizing pathways in the mouseS Huyghe, M Casteels, A Janssen, et al.
Nature Genetics|December 17, 1997
Urokinase-generated plasmin activates matrix metalloproteinases during aneurysm formationP Carmeliet, L Moons, R Lijnen, et al.
European Journal of Biochemistry|July 15, 1996
Rat pristanoyl-CoA oxidase. cDNA cloning and recognition of its C-terminal (SQL) by the peroxisomal-targeting signal 1 receptorJ C Vanhooren, M Fransen, B de Béthune, et al.
Nature Genetics|September 1, 1997
A mouse model for Zellweger syndromeM Baes, P Gressens, E Baumgart, et al.
Journal of Neuropathology and Experimental Neurology|April 10, 2002
The neuronal migration defect in mice with Zellweger syndrome (Pex5 knockout) is not caused by the inactivity of peroxisomal beta-oxidationM Baes, P Gressens, S Huyghe, et al.
Cellular and Molecular Life Sciences : CMLS|June 23, 2006
Farnesylation of Pex19p is not essential for peroxisome biogenesis in yeast and mammalian cellsI M K Vastiau, E A Anthonio, M Brams, et al.
Nature Medicine|September 30, 1999
Inhibition of plasminogen activators or matrix metalloproteinases prevents cardiac rupture but impairs therapeutic angiogenesis and causes cardiac failureS Heymans, A Luttun, D Nuyens, et al.
Pageof 4