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Biochemical and Biophysical Research Communications
|
June 28, 2005
Connexin43 with a cytoplasmic loop deletion inhibits the function of several connexins
Min Wang, Agustín D Martínez, Viviana M Berthoud, et al.
Experimental Eye Research
|
November 3, 2012
An MIP/AQP0 mutation with impaired trafficking and function underlies an autosomal dominant congenital lamellar cataract
G Senthil Kumar, John W Kyle, Peter J Minogue, et al.
Molecular Vision
|
September 17, 2009
The GJA8 allele encoding CX50I247M is a rare polymorphism, not a cataract-causing mutation
Jochen Graw, Werner Schmidt, Peter J Minogue, et al.
Journal of Proteome Research
|
December 17, 2010
Identification of CaMKII phosphorylation sites in Connexin43 by high-resolution mass spectrometry
Richard Y-C Huang, James G Laing, Evelyn M Kanter, et al.
Investigative Ophthalmology & Visual Science
|
August 18, 2009
A mutant connexin50 with enhanced hemichannel function leads to cell death
Peter J Minogue, Jun-Jie Tong, Anita Arora, et al.
Molecular Biosystems
|
January 6, 2012
Modulation of gap junction channels and hemichannels by growth factors
Kurt A Schalper, Manuel A Riquelme, María C Brañes, et al.
Biological Research
|
May 23, 2024
Connexin channels and hemichannels are modulated differently by charge reversal at residues forming the intracellular pocket
Felipe Villanelo, Peter J Minogue, Jaime Maripillán, et al.
Journal of Medical Genetics
|
November 17, 2007
A novel connexin50 mutation associated with congenital nuclear pulverulent cataracts
A Arora, P J Minogue, X Liu, et al.
Molecular Biology of the Cell
|
July 13, 2012
Critical role of the first transmembrane domain of Cx26 in regulating oligomerization and function
Oscar Jara, Rodrigo Acuña, Isaac E García, et al.
Journal of Medical Genetics
|
January 7, 2006
A novel GJA8 mutation is associated with autosomal dominant lamellar pulverulent cataract: further evidence for gap junction dysfunction in human cataract
A Arora, P J Minogue, X Liu, et al.
Page
of 11
Search research articles
Search
Showing results (91-100 of 102) with videos related to
Sort By:
Page
of 11
Biochemical and Biophysical Research Communications
|
June 28, 2005
Connexin43 with a cytoplasmic loop deletion inhibits the function of several connexins
Min Wang, Agustín D Martínez, Viviana M Berthoud, et al.
Experimental Eye Research
|
November 3, 2012
An MIP/AQP0 mutation with impaired trafficking and function underlies an autosomal dominant congenital lamellar cataract
G Senthil Kumar, John W Kyle, Peter J Minogue, et al.
Molecular Vision
|
September 17, 2009
The GJA8 allele encoding CX50I247M is a rare polymorphism, not a cataract-causing mutation
Jochen Graw, Werner Schmidt, Peter J Minogue, et al.
Journal of Proteome Research
|
December 17, 2010
Identification of CaMKII phosphorylation sites in Connexin43 by high-resolution mass spectrometry
Richard Y-C Huang, James G Laing, Evelyn M Kanter, et al.
Investigative Ophthalmology & Visual Science
|
August 18, 2009
A mutant connexin50 with enhanced hemichannel function leads to cell death
Peter J Minogue, Jun-Jie Tong, Anita Arora, et al.
Molecular Biosystems
|
January 6, 2012
Modulation of gap junction channels and hemichannels by growth factors
Kurt A Schalper, Manuel A Riquelme, María C Brañes, et al.
Biological Research
|
May 23, 2024
Connexin channels and hemichannels are modulated differently by charge reversal at residues forming the intracellular pocket
Felipe Villanelo, Peter J Minogue, Jaime Maripillán, et al.
Journal of Medical Genetics
|
November 17, 2007
A novel connexin50 mutation associated with congenital nuclear pulverulent cataracts
A Arora, P J Minogue, X Liu, et al.
Molecular Biology of the Cell
|
July 13, 2012
Critical role of the first transmembrane domain of Cx26 in regulating oligomerization and function
Oscar Jara, Rodrigo Acuña, Isaac E García, et al.
Journal of Medical Genetics
|
January 7, 2006
A novel GJA8 mutation is associated with autosomal dominant lamellar pulverulent cataract: further evidence for gap junction dysfunction in human cataract
A Arora, P J Minogue, X Liu, et al.
Page
of 11