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Journal of Cellular Biochemistry. Supplement|July 17, 2001
Nuclear changes in necrotic HL-60 cellsR Bortul, M Zweyer, A M Billi, et al.Cell Biology International Reports|May 1, 1988
Evidence for a reduction of the replicative activity of matrix-bound DNA polymerase alpha following treatment with phospholipase CS Miscia, A Cataldi, A Ognibene, et al.Cancer Research|June 1, 1997
Essential role for nuclear phospholipase C beta1 in insulin-like growth factor I-induced mitogenesisL Manzoli, A M Billi, S Rubbini, et al.Biochemical and Biophysical Research Communications|February 12, 1998
Control of expression of PLCbeta1 by LAC repressor system: relationship between nuclear PLCbeta1 overexpression and growth factor stimulationA M Billi, A Matteucci, I Faenza, et al.Cancer Research|July 15, 1995
Phosphoinositide signaling in nuclei of Friend cells: tiazofurin down-regulates phospholipase C beta 1L Manzoli, A M Billi, R S Gilmour, et al.Leukemia|August 22, 2003
The phosphoinositide 3-kinase/Akt pathway regulates cell cycle progression of HL60 human leukemia cells through cytoplasmic relocalization of the cyclin-dependent kinase inhibitor p27(Kip1) and control of cyclin D1 expressionA Cappellini, G Tabellini, M Zweyer, et al.Biochimica Et Biophysica Acta|April 13, 2000
Identification and chromosomal localisation by fluorescence in situ hybridisation of human gene of phosphoinositide-specific phospholipase C beta(1)D Peruzzi, G Calabrese, I Faenza, et al.Leukemia|February 20, 2003
Constitutively active Akt1 protects HL60 leukemia cells from TRAIL-induced apoptosis through a mechanism involving NF-kappaB activation and cFLIP(L) up-regulationR Bortul, P L Tazzari, A Cappellini, et al.Leukemia|September 13, 2003
A new selective AKT pharmacological inhibitor reduces resistance to chemotherapeutic drugs, TRAIL, all-trans-retinoic acid, and ionizing radiation of human leukemia cellsA M Martelli, P L Tazzari, G Tabellini, et al.Cancer Research|November 21, 1998
Nuclear but not cytoplasmic phospholipase C beta 1 inhibits differentiation of erythroleukemia cellsA Matteucci, I Faenza, R S Gilmour, et al.Pageof 5