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Immunology Today|June 4, 1999
Novel mechanism for the impairment of cell proliferation in HIV-1 infectionM Bofill, N J Borthwick, H A Simmonds
The Journal of Infectious Diseases|January 7, 1999
Activation-associated necrosis in human immunodeficiency virus infectionN J Borthwick, R G Wickremasinghe, J Lewin, et al.
Immunology|August 1, 1996
Factors that influence activated CD8+ T-cell apoptosis in patients with acute herpesvirus infections: loss of costimulatory molecules CD28, CD5 and CD6 but relative maintenance of Bax and Bcl-X expressionN J Borthwick, M Bofill, I Hassan, et al.
The Journal of Experimental Medicine|November 1, 1994
The specific recognition by macrophages of CD8+,CD45RO+ T cells undergoing apoptosis: a mechanism for T cell clearance during resolution of viral infectionsA N Akbar, J Savill, W Gombert, et al.
European Journal of Immunology|February 1, 1996
Interleukin-2 receptor common gamma-chain signaling cytokines regulate activated T cell apoptosis in response to growth factor withdrawal: selective induction of anti-apoptotic (bcl-2, bcl-xL) but not pro-apoptotic (bax, bcl-xS) gene expressionA N Akbar, N J Borthwick, R G Wickremasinghe, et al.
AIDS (London, England)|April 1, 1994
Lymphocyte activation in HIV-1 infection. II. Functional defects of CD28- T cellsN J Borthwick, M Bofill, W M Gombert, et al.
Immunology|November 1, 1996
Fibroblasts prevent apoptosis of IL-2-deprived T cells without inducing proliferation: a selective effect on Bcl-XL expressionW Gombert, N J Borthwick, D L Wallace, et al.
The American Journal of Pathology|June 1, 1995
Presence of CD3+CD8+Bcl-2(low) lymphocytes undergoing apoptosis and activated macrophages in lymph nodes of HIV-1+ patientsM Bofill, W Gombert, N J Borthwick, et al.
AIDS (London, England)|July 1, 1996
Increased numbers of primed activated CD8+CD38+CD45RO+ T cells predict the decline of CD4+ T cells in HIV-1-infected patientsM Bofill, A Mocroft, M Lipman, et al.
International Immunology|July 6, 2000
Loss of CD28 expression on CD8(+) T cells is induced by IL-2 receptor gamma chain signalling cytokines and type I IFN, and increases susceptibility to activation-induced apoptosisN J Borthwick, M Lowdell, M Salmon, et al.
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