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Journal of Neural Transmission. Supplementum|May 21, 1999
Myelin dysfunction/degradation in the central nervous system: why are myelin sheaths susceptible to damage?M BradlProceedings of the National Academy of Sciences of the United States of America|November 1, 1991
Mosaic expression of a tyrosinase fusion gene in albino mice yields a heritable striped coat color pattern in transgenic homozygotesB Mintz, M BradlGlia|October 12, 2001
New tools to trace populations of inflammatory cells in the CNSA Flügel, M BradlBrain Pathology (Zurich, Switzerland)|July 1, 1996
Animal models of demyelinationM Bradl, C LiningtonJournal of Neurology, Neurosurgery, and Psychiatry|October 23, 2003
Molecular pathogenesis of neuroinflammationM Bradl, R HohlfeldInternational MS Journal|September 25, 2008
Anti-aquaporin-4 antibodies in neuromyelitis optica: how to prove their pathogenetic relevance?M Bradl, Dr Hans LassmannJournal of Neuroimmunology|September 1, 1989
Clonotypic chromosomal aberrations in long-term lines of myelin-specific rat T lymphocytesM Bradl, M Schmid, H WekerleProceedings of the National Academy of Sciences of the United States of America|August 1, 1991
Clonal coat color variation due to a transforming gene expressed in melanocytes of transgenic miceM Bradl, L Larue, B MintzActa Neuropathologica|January 1, 1988
Experimental allergic encephalomyelitis: the balance between encephalitogenic T lymphocytes and demyelinating antibodies determines size and structure of demyelinated lesionsH Lassmann, C Brunner, M Bradl, et al.Oncogene|March 1, 1993
Melanocyte culture lines from Tyr-SV40E transgenic mice: models for the molecular genetic evolution of malignant melanomaL Larue, N Dougherty, M Bradl, et al.Pageof 2