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Brain Pathology (Zurich, Switzerland)|July 1, 1996
Animal models of demyelinationM Bradl, C LiningtonActa Neuropathologica|January 1, 1988
Experimental allergic encephalomyelitis: the balance between encephalitogenic T lymphocytes and demyelinating antibodies determines size and structure of demyelinated lesionsH Lassmann, C Brunner, M Bradl, et al.Immunological Reviews|February 1, 1996
The shaping of the brain-specific T lymphocyte repertoire in the thymusH Wekerle, M Bradl, C Linington, et al.The American Journal of Pathology|March 1, 1988
Augmentation of demyelination in rat acute allergic encephalomyelitis by circulating mouse monoclonal antibodies directed against a myelin/oligodendrocyte glycoproteinC Linington, M Bradl, H Lassmann, et al.The American Journal of Pathology|September 15, 1998
T-cell apoptosis in inflammatory brain lesions: destruction of T cells does not depend on antigen recognitionJ Bauer, M Bradl, W F Hickley, et al.Journal of Neural Transmission. Supplementum|May 21, 1999
Myelin dysfunction/degradation in the central nervous system: why are myelin sheaths susceptible to damage?M BradlProceedings of the National Academy of Sciences of the United States of America|November 1, 1991
Mosaic expression of a tyrosinase fusion gene in albino mice yields a heritable striped coat color pattern in transgenic homozygotesB Mintz, M BradlGlia|October 12, 2001
New tools to trace populations of inflammatory cells in the CNSA Flügel, M BradlJournal of Neurology, Neurosurgery, and Psychiatry|October 23, 2003
Molecular pathogenesis of neuroinflammationM Bradl, R HohlfeldNeurochemical Research|September 1, 1992
Molecular mimicry and the autoimmune response to the peripheral nerve myelin P0 glycoproteinM Adelmann, C LiningtonPageof 11