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Scandinavian Journal of Immunology|February 14, 2002
Fc receptors are critical for autoimmune inflammatory damage to the central nervous system in experimental autoimmune encephalomyelitisK-B Abdul-Majid, A Stefferl, C Bourquin, et al.
The American Journal of Pathology|July 6, 2000
Multiple sclerosis and chronic autoimmune encephalomyelitis: a comparative quantitative study of axonal injury in active, inactive, and remyelinated lesionsB Kornek, M K Storch, R Weissert, et al.
The Journal of Experimental Medicine|September 1, 1994
Experimental autoimmune panencephalitis and uveoretinitis transferred to the Lewis rat by T lymphocytes specific for the S100 beta molecule, a calcium binding protein of astrogliaK Kojima, T Berger, H Lassmann, et al.
Brain Pathology (Zurich, Switzerland)|November 6, 1998
Autoimmunity to myelin oligodendrocyte glycoprotein in rats mimics the spectrum of multiple sclerosis pathologyM K Storch, A Stefferl, U Brehm, et al.
Science (New York, N.Y.)|December 20, 1996
Late complications of immune deviation therapy in a nonhuman primateC P Genain, K Abel, N Belmar, et al.
European Journal of Immunology|June 1, 1993
T cells specific for the myelin oligodendrocyte glycoprotein mediate an unusual autoimmune inflammatory response in the central nervous systemC Linington, T Berger, L Perry, et al.
Journal of Neuroimmunology|December 1, 1995
The N-terminal domain of the myelin oligodendrocyte glycoprotein (MOG) induces acute demyelinating experimental autoimmune encephalomyelitis in the Lewis ratM Adelmann, J Wood, I Benzel, et al.
Human Molecular Genetics|November 5, 1999
Linkage analysis of myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitis in the rat identifies a locus controlling demyelination on chromosome 18I Dahlman, E Wallström, R Weissert, et al.
Experimental Neurology|June 28, 2012
Re-expression of N-cadherin in remyelinating lesions of experimental inflammatory demyelinationS Hochmeister, M Romauch, J Bauer, et al.
Endocrinology|July 19, 2001
Disease progression in chronic relapsing experimental allergic encephalomyelitis is associated with reduced inflammation-driven production of corticosteroneA Stefferl, M K Storch, C Linington, et al.
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