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M Brass

Showing results (81-90 of 198) with videos related to

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American Journal of Physiology. Lung Cellular and Molecular Physiology|June 24, 2003
Fibrinolysis in LPS-induced chronic airway diseaseJordan D Savov, David M Brass, Katherine G Berman, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|June 10, 2003
Subchronic endotoxin inhalation causes persistent airway diseaseD M Brass, J D Savov, S H Gavett, et al.
Annals of the New York Academy of Sciences|February 24, 2001
PACAP peptides modulate guinea pig cardiac neuron membrane excitability and neuropeptide expressionR L Parsons, T M Rossignol, M A Calupca, et al.
The American Journal of Pathology|March 18, 1999
Reduced tumor necrosis factor-alpha and transforming growth factor-beta1 expression in the lungs of inbred mice that fail to develop fibroproliferative lesions consequent to asbestos exposureD M Brass, G W Hoyle, H G Poovey, et al.
Annals of Internal Medicine|April 1, 1991
A prognostic system for transient ischemia or minor strokeW N Kernan, R I Horwitz, L M Brass, et al.
Stroke|June 28, 2008
Dependence in prestroke mobility predicts adverse outcomes among patients with acute ischemic strokeMary I Dallas, Shari Rone-Adams, John L Echternach, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|October 12, 2002
Neutrophils play a critical role in development of LPS-induced airway diseaseJordan D Savov, Stephen H Gavett, David M Brass, et al.
Journal of Clinical Epidemiology|February 11, 1999
Stratified randomization for clinical trialsW N Kernan, C M Viscoli, R W Makuch, et al.
The Journal of Allergy and Clinical Immunology|September 10, 2004
LPS binding protein is important in the airway response to inhaled endotoxinDavid M Brass, Jordan D Savov, Gregory S Whitehead, et al.
Clinical and Experimental Allergy : Journal of the British Society for Allergy and Clinical Immunology|June 20, 2008
Bakery flour dust exposure causes non-allergic inflammation and enhances allergic airway inflammation in miceP Marraccini, D M Brass, J W Hollingsworth, et al.
Pageof 20

Showing results (81-90 of 198) with videos related to

Sort By:
Pageof 20
American Journal of Physiology. Lung Cellular and Molecular Physiology|June 24, 2003
Fibrinolysis in LPS-induced chronic airway diseaseJordan D Savov, David M Brass, Katherine G Berman, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|June 10, 2003
Subchronic endotoxin inhalation causes persistent airway diseaseD M Brass, J D Savov, S H Gavett, et al.
Annals of the New York Academy of Sciences|February 24, 2001
PACAP peptides modulate guinea pig cardiac neuron membrane excitability and neuropeptide expressionR L Parsons, T M Rossignol, M A Calupca, et al.
The American Journal of Pathology|March 18, 1999
Reduced tumor necrosis factor-alpha and transforming growth factor-beta1 expression in the lungs of inbred mice that fail to develop fibroproliferative lesions consequent to asbestos exposureD M Brass, G W Hoyle, H G Poovey, et al.
Annals of Internal Medicine|April 1, 1991
A prognostic system for transient ischemia or minor strokeW N Kernan, R I Horwitz, L M Brass, et al.
Stroke|June 28, 2008
Dependence in prestroke mobility predicts adverse outcomes among patients with acute ischemic strokeMary I Dallas, Shari Rone-Adams, John L Echternach, et al.
American Journal of Physiology. Lung Cellular and Molecular Physiology|October 12, 2002
Neutrophils play a critical role in development of LPS-induced airway diseaseJordan D Savov, Stephen H Gavett, David M Brass, et al.
Journal of Clinical Epidemiology|February 11, 1999
Stratified randomization for clinical trialsW N Kernan, C M Viscoli, R W Makuch, et al.
The Journal of Allergy and Clinical Immunology|September 10, 2004
LPS binding protein is important in the airway response to inhaled endotoxinDavid M Brass, Jordan D Savov, Gregory S Whitehead, et al.
Clinical and Experimental Allergy : Journal of the British Society for Allergy and Clinical Immunology|June 20, 2008
Bakery flour dust exposure causes non-allergic inflammation and enhances allergic airway inflammation in miceP Marraccini, D M Brass, J W Hollingsworth, et al.
Pageof 20