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Tissue Antigens|November 5, 1998
HLA-Cw alleles associated with HLA extended haplotypes and C2 deficiencyO P Clavijo, J C Delgado, Z L Awdeh, et al.
The Journal of Clinical Investigation|December 1, 1991
A restriction fragment of the C2 gene is a unique marker for C2 deficiency and the uncommon C2 allele C2*B (a marker for type 1 diabetes)S Simon, Z Awdeh, R D Campbell, et al.
The Journal of Clinical Investigation|June 1, 1994
Two subsets of HLA-DQA1 alleles mark phenotypic variation in levels of insulin autoantibodies in first degree relatives at risk for insulin-dependent diabetesA Pugliese, T Bugawan, R Moromisato, et al.
Diabetes|June 1, 1995
HLA-DQB1*0602 is associated with dominant protection from diabetes even among islet cell antibody-positive first-degree relatives of patients with IDDMA Pugliese, R Gianani, R Moromisato, et al.
The Journal of Biological Chemistry|February 25, 1992
Covalent binding of C3b to C4b within the classical complement pathway C5 convertase. Determination of amino acid residues involved in ester linkage formationY U Kim, M C Carroll, D E Isenman, et al.
The Journal of Experimental Medicine|May 1, 1996
Reperfusion injury of ischemic skeletal muscle is mediated by natural antibody and complementM R Weiser, J P Williams, F D Moore, et al.
The Journal of Experimental Medicine|December 1, 1993
Major histocompatibility complex susceptibility genes for dermatitis herpetiformis compared with those for gluten-sensitive enteropathyA R Ahmed, J J Yunis, D Marcus-Bagley, et al.
Immunogenetics|January 1, 1984
Linkage disequilibrium of HLA-SB1 with the HLA-A1, B8, DR3, SCO1 and of HLA-SB4 with the HLA-A26, Bw38, Dw10, DR4, SC21 extended haplotypesY Matsui, S M Alosco, Z Awdeh, et al.
Diabetes|June 1, 1991
Specific association of HLA-DR4 with increased prevalence and level of insulin autoantibodies in first-degree relatives of patients with type I diabetesR Ziegler, C A Alper, Z L Awdeh, et al.
Journal of Immunology (Baltimore, Md. : 1950)|December 20, 2000
A role for CD21/CD35 and CD19 in responses to acute septic peritonitis: a potential mechanism for mast cell activationJ L Gommerman, D Y Oh, X Zhou, et al.
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