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Blood|December 15, 1990
Restoration of phagocyte function by interferon-gamma in X-linked chronic granulomatous disease occurs at the level of a progenitor cellR A Ezekowitz, C A Sieff, M C Dinauer, et al.Proceedings of the National Academy of Sciences of the United States of America|May 1, 1988
Primary structure and unique expression of the 22-kilodalton light chain of human neutrophil cytochrome bC A Parkos, M C Dinauer, L E Walker, et al.Critical Reviews in Clinical Laboratory Sciences|January 1, 1993
The respiratory burst oxidase and the molecular genetics of chronic granulomatous diseaseM C DinauerCurrent Opinion in Pediatrics|February 1, 1993
Leukocyte function and nonmalignant leukocyte disordersM C DinauerBlood|September 15, 1992
Splice site mutations are a common cause of X-linked chronic granulomatous diseaseM de Boer, B G Bolscher, M C Dinauer, et al.Proceedings of the National Academy of Sciences of the United States of America|December 15, 1991
Point mutation in the cytoplasmic domain of the neutrophil p22-phox cytochrome b subunit is associated with a nonfunctional NADPH oxidase and chronic granulomatous diseaseM C Dinauer, E A Pierce, R W Erickson, et al.The Journal of Biological Chemistry|August 25, 1977
In vitro synthesis of a DNA probe for antisense globin sequencesS H OrkinThe Journal of Biological Chemistry|January 10, 1978
Selective restriction endonuclease cleavage of human globin genesS H OrkinIn Vitro|January 1, 1978
Differentiation of murine erythroleukemic (Friend) cells: an in vitro model of erythropoiesisS H OrkinPageof 31