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Neurology|January 21, 2009
Plasma Abeta, homocysteine, and cognition: the Vitamin Intervention for Stroke Prevention (VISP) trialA Viswanathan, S Raj, S M Greenberg, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 15, 1997
Abeta deposition is associated with neuropil changes, but not with overt neuronal loss in the human amyloid precursor protein V717F (PDAPP) transgenic mouseM C Irizarry, F Soriano, M McNamara, et al.
Acta Neuropathologica|October 25, 2000
Apolipoprotein E affects the amount, form, and anatomical distribution of amyloid beta-peptide deposition in homozygous APP(V717F) transgenic miceM C Irizarry, B S Cheung, G W Rebeck, et al.
Annals of the New York Academy of Sciences|February 24, 2001
Modulation of A beta deposition in APP transgenic mice by an apolipoprotein E null backgroundM C Irizarry, G W Rebeck, B Cheung, et al.
Biophysical Journal|February 27, 1999
Dynamics of plaque formation in Alzheimer's diseaseB Urbanc, L Cruz, S V Buldyrev, et al.
Journal of Neuropathology and Experimental Neurology|May 26, 1998
Nigral and cortical Lewy bodies and dystrophic nigral neurites in Parkinson's disease and cortical Lewy body disease contain alpha-synuclein immunoreactivityM C Irizarry, W Growdon, T Gomez-Isla, et al.
Journal of Neuropathology and Experimental Neurology|August 1, 1996
Characterization of the precursor protein of the non-A beta component of senile plaques (NACP) in the human central nervous systemM C Irizarry, T W Kim, M McNamara, et al.
The Journal of Neuropsychiatry and Clinical Neurosciences|August 10, 1999
Risperidone treatment of behavioral disturbances in outpatients with dementiaM C Irizarry, S N Ghaemi, E R Lee-Cherry, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 11, 2002
Neurotoxic effects of thioflavin S-positive amyloid deposits in transgenic mice and Alzheimer's diseaseB Urbanc, L Cruz, R Le, et al.
Journal of Neuropathology and Experimental Neurology|August 7, 2001
Plaque-induced abnormalities in neurite geometry in transgenic models of Alzheimer disease: implications for neural system disruptionR Le, L Cruz, B Urbanc, et al.
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