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Annals of the New York Academy of Sciences|December 16, 1996
Epidemiological, clinical, and neuropathological study of apolipoprotein E genotype in Alzheimer's diseaseB T Hyman, T Gomez-Isla, G W Rebeck, et al.Annals of Neurology|January 1, 1996
Clinical and pathological correlates of apolipoprotein E epsilon 4 in Alzheimer's diseaseT Gomez-Isla, H L West, G W Rebeck, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|October 1, 1993
Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acidM F Beal, E Brouillet, B G Jenkins, et al.Journal of Neurochemistry|July 19, 2000
Aspartate mutations in presenilin and gamma-secretase inhibitors both impair notch1 proteolysis and nuclear translocation with relative preservation of notch1 signalingO Berezovska, C Jack, P McLean, et al.Annals of Neurology|April 1, 1995
Cerebrospinal fluid levels of amyloid beta-protein in Alzheimer's disease: inverse correlation with severity of dementia and effect of apolipoprotein E genotypeR M Nitsch, G W Rebeck, M Deng, et al.Nature Communications|June 2, 2016
Early detection of cryptic memory and glucose uptake deficits in pre-pathological APP miceV Beglopoulos, J Tulloch, A D Roe, et al.AJNR. American Journal of Neuroradiology|September 15, 2012
Apolipoprotein E epsilon4 does not modulate amyloid-β-associated neurodegeneration in preclinical Alzheimer diseaseR S Desikan, L K McEvoy, D Holland, et al.Brain : a Journal of Neurology|September 1, 1999
The impact of different presenilin 1 andpresenilin 2 mutations on amyloid deposition, neurofibrillary changes and neuronal loss in the familial Alzheimer's disease brain: evidence for other phenotype-modifying factorsT Gómez-Isla, W B Growdon, M J McNamara, et al.Annals of Neurology|February 24, 2001
No evidence for genetic association or linkage of the cathepsin D (CTSD) exon 2 polymorphism and Alzheimer diseaseL Bertram, S Guénette, J Jones, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|September 15, 1997
Mechanisms of reduced striatal NMDA excitotoxicity in type I nitric oxide synthase knock-out miceC Ayata, G Ayata, H Hara, et al.Pageof 24