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Journal of Neuropathology and Experimental Neurology|November 1, 1993
The lack of accumulation of senile plaques or amyloid burden in Alzheimer's disease suggests a dynamic balance between amyloid deposition and resolutionB T Hyman, K Marzloff, P V Arriagada
Trends in Neurosciences|April 1, 1993
Do defects in mitochondrial energy metabolism underlie the pathology of neurodegenerative diseases?M F Beal, B T Hyman, W Koroshetz
Journal of Neurology, Neurosurgery, and Psychiatry|November 1, 1985
Effect of naltrexone on senile dementia of the Alzheimer typeB T Hyman, P J Eslinger, A R Damasio
Biochimica Et Biophysica Acta|November 12, 1982
Prostaglandin production by 3T3-L1 cells in cultureB T Hyman, L L Stoll, A A Spector
Journal of Neuropathology and Experimental Neurology|January 1, 1995
Functional alterations in Alzheimer's disease: decreased glucose transporter 3 immunoreactivity in the perforant pathway terminal zoneS D Harr, N A Simonian, B T Hyman
Archives of Neurology|July 25, 1998
Apolipoprotein E genotype and deposits of Abeta40 and Abeta42 in Alzheimer diseaseM J McNamara, T Gomez-Isla, B T Hyman
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