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Journal of Virology|March 1, 1984
Common and distinct regions of defective-interfering RNAs of Sindbis virusS S Monroe, S SchlesingerDeutsche Zahnarztliche Zeitschrift|January 1, 1980
[Quantitative shifting of intra and peritumoral cell populations after intratumoral BCG-cell-wall therapy in squamous epithelial mouth neoplasms]S Schlesinger, H Pickartz, J BierThe Journal of Clinical Investigation|April 1, 1990
Phagocytosis of leprosy bacilli is mediated by complement receptors CR1 and CR3 on human monocytes and complement component C3 in serumL S Schlesinger, M A HorwitzInfection and Immunity|January 1, 1994
A role for natural antibody in the pathogenesis of leprosy: antibody in nonimmune serum mediates C3 fixation to the Mycobacterium leprae surface and hence phagocytosis by human mononuclear phagocytesL S Schlesinger, M A HorwitzJournal of Maxillofacial Surgery|December 1, 1983
The iliac bone or osteocutaneous transplant pedicled to the deep circumflex iliac artery. II. Clinical applicationK Bitter, S Schlesinger, U WestermanThe Journal of Experimental Medicine|November 1, 1991
Phenolic glycolipid-1 of Mycobacterium leprae binds complement component C3 in serum and mediates phagocytosis by human monocytesL S Schlesinger, M A HorwitzDiabetes Research and Clinical Practice|August 2, 2014
Impact of diabetes on the natural history of tuberculosisBlanca I Restrepo, Larry S SchlesingerProceedings of the National Academy of Sciences of the United States of America|July 1, 1987
Engineered defective interfering RNAs of Sindbis virus express bacterial chloramphenicol acetyltransferase in avian cellsR Levis, H Huang, S SchlesingerJournal of Virology|April 1, 1997
Sindbis virus replicons and Sindbis virus: assembly of chimeras and of particles deficient in virus RNAI Frolov, E Frolova, S SchlesingerJournal of Immunology (Baltimore, Md. : 1950)|September 15, 1991
Phagocytosis of Mycobacterium leprae by human monocyte-derived macrophages is mediated by complement receptors CR1 (CD35), CR3 (CD11b/CD18), and CR4 (CD11c/CD18) and IFN-gamma activation inhibits complement receptor function and phagocytosis of this bacteriumL S Schlesinger, M A HorwitzPageof 32