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Molecular and Cellular Pediatrics|February 8, 2018
The potential of antisense oligonucleotide therapies for inherited childhood lung diseasesKelly M Martinovich, Nicole C Shaw, Anthony Kicic, et al.Frontiers in Genetics|January 21, 2020
Consequences of Making the Inactive Active Through Changes in Antisense Oligonucleotide ChemistriesKhine Zaw, Kane Greer, May Thandar Aung-Htut, et al.Canadian Medical Association Journal|June 10, 1972
Arbovirus infections in man in British ColumbiaG D Kettyls, V M Verrall, L D Wilton, et al.Scientific Reports|April 23, 2020
Splice modulating antisense oligonucleotides restore some acid-alpha-glucosidase activity in cells derived from patients with late-onset Pompe diseaseMay Thandar Aung-Htut, Kristin A Ham, Michel Tchan, et al.International Journal of Clinical and Experimental Pathology|September 17, 2013
Investigation of splicing changes and post-translational processing of LMNA in sporadic inclusion body myositisYue-Bei Luo, Chalermchai Mitrpant, Russell Johnsen, et al.International Journal of Clinical and Experimental Pathology|December 3, 2013
Investigation of age-related changes in LMNA splicing and expression of progerin in human skeletal musclesYue-Bei Luo, Chalermchai Mitrpant, Russell D Johnsen, et al.Cytogenetics and Cell Genetics|January 1, 1995
Assignment of the human a-tropomyosin gene TPM3 to 1q22-->q23 by fluorescence in situ hybridisationS D Wilton, H Eyre, P A Akkari, et al.The Journal of Cell Biology|March 8, 2000
Massive idiosyncratic exon skipping corrects the nonsense mutation in dystrophic mouse muscle and produces functional revertant fibers by clonal expansionQ L Lu, G E Morris, S D Wilton, et al.Cytogenetics and Cell Genetics|January 1, 1993
Assignment of the human slow skeletal muscle troponin gene (TNNI1) to 1q32 by fluorescence in situ hybridisationH J Eyre, P A Akkari, C Meredith, et al.Plos One|June 4, 2014
Antisense oligonucleotide induction of progerin in human myogenic cellsYue-Bei Luo, Chalermchai Mitrpant, Abbie M Adams, et al.Pageof 24