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Molecular Therapy : the Journal of the American Society of Gene Therapy|December 14, 2006
The influence of antisense oligonucleotide length on dystrophin exon skippingP L Harding, A M Fall, K Honeyman, et al.
Frontiers in Molecular Neuroscience|August 8, 2024
Down syndrome and DYRK1A overexpression: relationships and future therapeutic directionsAidan J Murphy, Steve D Wilton, May T Aung-Htut, et al.
Current Opinion in Lipidology|October 15, 2021
Splice correction therapies for familial hypercholesterolemic patients with low-density lipoprotein receptor mutationsCraig S McIntosh, Gerald F Watts, Steve D Wilton, et al.
Molecular Therapy. Nucleic Acids|November 24, 2020
Morpholino Oligomer-Induced Dystrophin Isoforms to Map the Functional Domains in the Dystrophin ProteinDunhui Li, Abbie M Adams, Russell D Johnsen, et al.
Plos One|October 19, 2017
Functional improvement of dystrophic muscle by repression of utrophin: let-7c interactionManoj K Mishra, Emanuele Loro, Kasturi Sengupta, et al.
Gene Therapy|May 26, 2006
Antisense oligonucleotide-induced exon skipping restores dystrophin expression in vitro in a canine model of DMDG McClorey, H M Moulton, P L Iversen, et al.
International Journal of Molecular Sciences|July 8, 2020
Single Exon Skipping Can Address a Multi-Exon Duplication in the Dystrophin GeneKane Greer, Russell Johnsen, Yoram Nevo, et al.
Nature Biotechnology|April 29, 1998
Induction of primary carcinoembryonic antigen (CEA)-specific cytotoxic T lymphocytes in vitro using human dendritic cells transfected with RNAS K Nair, D Boczkowski, M Morse, et al.
Elife|June 9, 2021
Calcium signaling through a transient receptor channel is important for Toxoplasma gondii growthKarla Marie Márquez-Nogueras, Miryam Andrea Hortua Triana, Nathan M Chasen, et al.
Molecular Therapy. Nucleic Acids|December 17, 2017
Rational Design of Short Locked Nucleic Acid-Modified 2'-O-Methyl Antisense Oligonucleotides for Efficient Exon-Skipping In VitroBao T Le, Abbie M Adams, Susan Fletcher, et al.
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