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Proceedings of the National Academy of Sciences of the United States of America|July 8, 1999
Transcriptional and posttranslational plasticity and the generation of inflammatory painC J Woolf, M CostiganThe Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 7, 2001
Developmental expression of the TTX-resistant voltage-gated sodium channels Nav1.8 (SNS) and Nav1.9 (SNS2) in primary sensory neuronsS C Benn, M Costigan, S Tate, et al.Molecular and Cellular Neurosciences|June 14, 2000
Diversity of expression of the sensory neuron-specific TTX-resistant voltage-gated sodium ion channels SNS and SNS2F Amaya, I Decosterd, T A Samad, et al.Drugs|January 1, 1994
A new strategy for the treatment of inflammatory pain. Prevention or elimination of central sensitizationC J WoolfNeuroscience Letters|March 23, 1984
A selective effect of naloxone on heterosynaptic C-fibre-mediated inhibitions in the rat dorsal hornC J WoolfNature|December 15, 1983
Evidence for a central component of post-injury pain hypersensitivityC J WoolfBrain Research|March 30, 1981
Intrathecal high dose morphine produces hyperalgesia in the ratC J WoolfPhilosophical Transactions of the Royal Society of London. Series B, Biological Sciences|March 29, 1996
Phenotypic modification of primary sensory neurons: the role of nerve growth factor in the production of persistent painC J WoolfPageof 19