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International Journal of Molecular Sciences|July 2, 2021
Molecular Mechanisms of the Deregulation of Muscle Contraction Induced by the R90P Mutation in Tpm3.12 and the Weakening of This Effect by BDM and W7Yurii S Borovikov, Daria D Andreeva, Stanislava V Avrova, et al.Archives of Biochemistry and Biophysics|December 31, 2013
Gly126Arg substitution causes anomalous behaviour of α-skeletal and β-smooth tropomyosins during the ATPase cycleNikita A Rysev, Ilya A Nevzorov, Stanislava V Avrova, et al.Catheterization and Cardiovascular Diagnosis|January 1, 1977
Left ventricular endomyocardial biopsy. III: ultrastructural characteristics of cardiomyopathy and cardiac hypertrophy with good or poor ventricular functionM J Davies, S Kennedy, I A Brooksby, et al.International Journal of Molecular Sciences|December 15, 2018
The Primary Causes of Muscle Dysfunction Associated with the Point Mutations in Tpm3.12; Conformational Analysis of Mutant Proteins as a Tool for Classification of MyopathiesYurii S Borovikov, Olga E Karpicheva, Armen O Simonyan, et al.Biochemical and Biophysical Research Communications|May 25, 2018
The reason for the low Ca2+-sensitivity of thin filaments associated with the Glu41Lys mutation in the TPM2 gene is "freezing" of tropomyosin near the outer domain of actin and inhibition of actin monomer switching off during the ATPase cycleStanislava V Avrova, Olga E Karpicheva, Nikita A Rysev, et al.American Heart Journal|June 1, 1993
Regional sympathetic innervation of the heart by means of metaiodobenzylguanidine imaging in silent ischemiaC F Shakespeare, C J Page, M J O'Doherty, et al.Lancet (London, England)|June 30, 1990
Association of antibodies against phospholipids with heart valve disease in systemic lupus erythematosusM A Khamashta, R Cervera, R A Asherson, et al.Studies in Health Technology and Informatics|September 7, 2011
Policy brief on the current status of certification of electronic Health Records in the US and EuropeGeorges De Moor, John O'Brien, Doug Fridsma, et al.Biochemical and Biophysical Research Communications|November 4, 2017
The reason for a high Ca2+-sensitivity associated with Arg91Gly substitution in TPM2 gene is the abnormal behavior and high flexibility of tropomyosin during the ATPase cycleYurii S Borovikov, Armen O Simonyan, Olga E Karpicheva, et al.Experimental Cell Research|September 8, 2004
Suppression of MeCP2beta expression inhibits neurite extension in PC12 cellsSorcha M Cusack, Troy T Rohn, Ryan J Medeck, et al.Pageof 23