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M DiFiglia

Showing results (51-60 of 71) with videos related to

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The Journal of Biological Chemistry|December 5, 2000
Pro-caspase-8 is predominantly localized in mitochondria and released into cytoplasm upon apoptotic stimulationZ H Qin, Y Wang, K K Kikly, et al.
Experimental Neurology|July 31, 1998
Wild-type and mutant huntingtins function in vesicle trafficking in the secretory and endocytic pathwaysJ Velier, M Kim, C Schwarz, et al.
Neuroscience|March 1, 1995
Quinolinic acid-induced increases in calbindin D28k immunoreactivity in rat striatal neurons in vivo and in vitro mimic the pattern seen in Huntington's diseaseQ Huang, D Zhou, E Sapp, et al.
Science (New York, N.Y.)|September 26, 1997
Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brainM DiFiglia, E Sapp, K O Chase, et al.
Neuroscience|January 1, 1995
Evidence for a preferential loss of enkephalin immunoreactivity in the external globus pallidus in low grade Huntington's disease using high resolution image analysisE Sapp, P Ge, H Aizawa, et al.
Neuroreport|July 7, 1997
Fast transport and retrograde movement of huntingtin and HAP 1 in axonsJ Block-Galarza, K O Chase, E Sapp, et al.
Annals of Neurology|October 24, 1997
Huntingtin localization in brains of normal and Huntington's disease patientsE Sapp, C Schwarz, K Chase, et al.
Journal of Neuropathology and Experimental Neurology|March 29, 2001
Early and progressive accumulation of reactive microglia in the Huntington disease brainE Sapp, K B Kegel, N Aronin, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 25, 2001
Caspase 3-cleaved N-terminal fragments of wild-type and mutant huntingtin are present in normal and Huntington's disease brains, associate with membranes, and undergo calpain-dependent proteolysisY J Kim, Y Yi, E Sapp, et al.
Neuroscience|June 11, 1999
Forskolin and dopamine D1 receptor activation increase huntingtin's association with endosomes in immortalized neuronal cells of striatal originM Kim, J Velier, K Chase, et al.
Pageof 8

Showing results (51-60 of 71) with videos related to

Sort By:
Pageof 8
The Journal of Biological Chemistry|December 5, 2000
Pro-caspase-8 is predominantly localized in mitochondria and released into cytoplasm upon apoptotic stimulationZ H Qin, Y Wang, K K Kikly, et al.
Experimental Neurology|July 31, 1998
Wild-type and mutant huntingtins function in vesicle trafficking in the secretory and endocytic pathwaysJ Velier, M Kim, C Schwarz, et al.
Neuroscience|March 1, 1995
Quinolinic acid-induced increases in calbindin D28k immunoreactivity in rat striatal neurons in vivo and in vitro mimic the pattern seen in Huntington's diseaseQ Huang, D Zhou, E Sapp, et al.
Science (New York, N.Y.)|September 26, 1997
Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brainM DiFiglia, E Sapp, K O Chase, et al.
Neuroscience|January 1, 1995
Evidence for a preferential loss of enkephalin immunoreactivity in the external globus pallidus in low grade Huntington's disease using high resolution image analysisE Sapp, P Ge, H Aizawa, et al.
Neuroreport|July 7, 1997
Fast transport and retrograde movement of huntingtin and HAP 1 in axonsJ Block-Galarza, K O Chase, E Sapp, et al.
Annals of Neurology|October 24, 1997
Huntingtin localization in brains of normal and Huntington's disease patientsE Sapp, C Schwarz, K Chase, et al.
Journal of Neuropathology and Experimental Neurology|March 29, 2001
Early and progressive accumulation of reactive microglia in the Huntington disease brainE Sapp, K B Kegel, N Aronin, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 25, 2001
Caspase 3-cleaved N-terminal fragments of wild-type and mutant huntingtin are present in normal and Huntington's disease brains, associate with membranes, and undergo calpain-dependent proteolysisY J Kim, Y Yi, E Sapp, et al.
Neuroscience|June 11, 1999
Forskolin and dopamine D1 receptor activation increase huntingtin's association with endosomes in immortalized neuronal cells of striatal originM Kim, J Velier, K Chase, et al.
Pageof 8