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M F Jobling

Showing results (1-10 of 8) with videos related to

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Journal of Neurochemistry|March 10, 2001
Homocysteine potentiates copper- and amyloid beta peptide-mediated toxicity in primary neuronal cultures: possible risk factors in the Alzheimer's-type neurodegenerative pathwaysA R White, X Huang, M F Jobling, et al.
The Journal of Membrane Biology|July 25, 2003
Copper modulation of ion channels of PrP[106-126] mutant prion peptide fragmentsJ I Kourie, B L Kenna, D Tew, et al.
The American Journal of Pathology|November 7, 1999
Prion protein-deficient neurons reveal lower glutathione reductase activity and increased susceptibility to hydrogen peroxide toxicityA R White, S J Collins, F Maher, et al.
Biochemistry|March 17, 1999
Familial prion disease mutation alters the secondary structure of recombinant mouse prion protein: implications for the mechanism of prion formationR Cappai, L Stewart, M F Jobling, et al.
Journal of Neuroscience Research|September 11, 2001
Involvement of the 5-lipoxygenase pathway in the neurotoxicity of the prion peptide PrP106-126L R Stewart, A R White, M F Jobling, et al.
Neurobiology of Disease|April 13, 2001
Sublethal concentrations of prion peptide PrP106-126 or the amyloid beta peptide of Alzheimer's disease activates expression of proapoptotic markers in primary cortical neuronsA R White, R Guirguis, M W Brazier, et al.
Journal of Neurochemistry|September 29, 1999
The hydrophobic core sequence modulates the neurotoxic and secondary structure properties of the prion peptide 106-126M F Jobling, L R Stewart, A R White, et al.
Biochemistry|July 4, 2001
Copper and zinc binding modulates the aggregation and neurotoxic properties of the prion peptide PrP106-126M F Jobling, X Huang, L R Stewart, et al.
Pageof 1

Showing results (1-10 of 8) with videos related to

Sort By:
Pageof 1
Journal of Neurochemistry|March 10, 2001
Homocysteine potentiates copper- and amyloid beta peptide-mediated toxicity in primary neuronal cultures: possible risk factors in the Alzheimer's-type neurodegenerative pathwaysA R White, X Huang, M F Jobling, et al.
The Journal of Membrane Biology|July 25, 2003
Copper modulation of ion channels of PrP[106-126] mutant prion peptide fragmentsJ I Kourie, B L Kenna, D Tew, et al.
The American Journal of Pathology|November 7, 1999
Prion protein-deficient neurons reveal lower glutathione reductase activity and increased susceptibility to hydrogen peroxide toxicityA R White, S J Collins, F Maher, et al.
Biochemistry|March 17, 1999
Familial prion disease mutation alters the secondary structure of recombinant mouse prion protein: implications for the mechanism of prion formationR Cappai, L Stewart, M F Jobling, et al.
Journal of Neuroscience Research|September 11, 2001
Involvement of the 5-lipoxygenase pathway in the neurotoxicity of the prion peptide PrP106-126L R Stewart, A R White, M F Jobling, et al.
Neurobiology of Disease|April 13, 2001
Sublethal concentrations of prion peptide PrP106-126 or the amyloid beta peptide of Alzheimer's disease activates expression of proapoptotic markers in primary cortical neuronsA R White, R Guirguis, M W Brazier, et al.
Journal of Neurochemistry|September 29, 1999
The hydrophobic core sequence modulates the neurotoxic and secondary structure properties of the prion peptide 106-126M F Jobling, L R Stewart, A R White, et al.
Biochemistry|July 4, 2001
Copper and zinc binding modulates the aggregation and neurotoxic properties of the prion peptide PrP106-126M F Jobling, X Huang, L R Stewart, et al.
Pageof 1