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Diabetes|February 23, 2022
ATP-Sensitive Potassium Channels in Hyperinsulinism and Type 2 Diabetes: Inconvenient Paradox or New Paradigm?Colin G Nichols, Nathaniel W York, Maria S RemediBiophysical Journal|February 13, 2001
ATP interaction with the open state of the K(ATP) channelD Enkvetchakul, G Loussouarn, E Makhina, et al.The Journal of Physiology|June 1, 1989
The mechanism of early contractile failure of isolated rat ventricular myocytes subjected to complete metabolic inhibitionW J Lederer, C G Nichols, G L SmithJournal of Applied Physiology (Bethesda, Md. : 1985)|March 10, 2000
Effects of loaded breathing and hypoxia on diaphragm metabolism as measured by (31)P-NMR spectroscopyP J Radell, S M Eleff, D G NicholsThe Journal of General Physiology|December 14, 2016
Diabetes induced by gain-of-function mutations in the Kir6.1 subunit of the KATP channelMaria S Remedi, Jonathan B Friedman, Colin G NicholsInternational Journal of Phytoremediation|August 10, 2019
Productivity and cost-effectiveness of short-rotation hardwoods on various land types in the southeastern USASolomon B Ghezehei, Elizabeth G Nichols, Dennis W HazelThe Journal of General Physiology|April 12, 2006
The polyamine binding site in inward rectifier K+ channelsHarley T Kurata, Laurence J Marton, Colin G NicholsPlant Physiology|April 1, 1987
Regulation of climacteric respiration in ripening avocado fruitA B Bennett, G M Smith, B G NicholsDiabetes|October 27, 2005
Diabetes and insulin secretion: the ATP-sensitive K+ channel (K ATP) connectionJoseph C Koster, M Alan Permutt, Colin G NicholsThe Journal of General Physiology|February 11, 2009
KirBac1.1: it's an inward rectifying potassium channelWayland W L Cheng, Decha Enkvetchakul, Colin G NicholsPageof 60