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JCI Insight|November 10, 2020
Kir6.1- and SUR2-dependent KATP overactivity disrupts intestinal motility in murine models of Cantú syndromeNathaniel W York, Helen Parker, Zili Xie, et al.
American Journal of Physiology. Heart and Circulatory Physiology|August 25, 2009
Impaired contractile function and calcium handling in hearts of cardiac-specific calcineurin b1-deficient micePaul J Schaeffer, Jaime Desantiago, John Yang, et al.
Journal of Immunology (Baltimore, Md. : 1950)|March 12, 2014
Metabolic reprogramming is required for antibody production that is suppressed in anergic but exaggerated in chronically BAFF-exposed B cellsAlfredo Caro-Maldonado, Ruoning Wang, Amanda G Nichols, et al.
Journal of Pediatric Surgery|December 1, 1989
Prediction of successful primary closure of congenital abdominal wall defects using intraoperative measurementsM Yaster, T L Scherer, M M Stone, et al.
Proceedings of the National Academy of Sciences of the United States of America|October 7, 2009
Dual role of K ATP channel C-terminal motif in membrane targeting and metabolic regulationCrystal F Kline, Harley T Kurata, Thomas J Hund, et al.
Function (Oxford, England)|September 1, 2020
The Mechanism of High-Output Cardiac Hypertrophy Arising From Potassium Channel Gain-of-Function in Cantú SyndromeConor McClenaghan, Yan Huang, Scot J Matkovich, et al.
The Journal of Biological Chemistry|November 22, 2013
Control of KirBac3.1 potassium channel gating at the interface between cytoplasmic domainsLejla Zubcevic, Vassiliy N Bavro, Joao R C Muniz, et al.
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