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Clinical and Diagnostic Virology|September 19, 1998
Repression of the PKR protein kinase by the hepatitis C virus NS5A protein: a potential mechanism of interferon resistanceM J Gale, M J Korth, M G Katze
Virology|March 1, 1996
Development of an in vitro mRNA degradation assay utilizing extracts from HIV-1- and SIV-infected cellsM B Agy, C H Sherbert, M G Katze
Virology|August 26, 1998
Interferon inhibits the replication of HIV-1, SIV, and SHIV chimeric viruses by distinct mechanismsM J Korth, M D Taylor, M G Katze
Virology|February 28, 1998
Interferon treatment inhibits the replication of simian immunodeficiency virus at an early stage: evidence for a block between attachment and reverse transcriptionM D Taylor, M J Korth, M G Katze
Proceedings of the National Academy of Sciences of the United States of America|August 15, 1991
Binding of the adenovirus VAI RNA to the interferon-induced 68-kDa protein kinase correlates with functionG D Ghadge, S Swaminathan, M G Katze, et al.
European Journal of Biochemistry|January 2, 1989
The binding of double-stranded RNA and adenovirus VAI RNA to the interferon-induced protein kinaseJ Galabru, M G Katze, N Robert, et al.
Journal of Virology|May 1, 1989
The cellular 68,000-Mr protein kinase is highly autophosphorylated and activated yet significantly degraded during poliovirus infection: implications for translational regulationT L Black, B Safer, A Hovanessian, et al.
Virology|July 1, 1990
Expression of cellular genes in CD4 positive lymphoid cells infected by the human immunodeficiency virus, HIV-1: evidence for a host protein synthesis shut-off induced by cellular mRNA degradationM B Agy, M Wambach, K Foy, et al.
Journal of Virology|April 1, 1983
Control of adenovirus gene expression: cellular gene products restrict expression of adenovirus host range mutants in nonpermissive cellsM G Katze, H Persson, B M Johansson, et al.
Virology|December 1, 1995
Binding of the influenza virus NS1 protein to double-stranded RNA inhibits the activation of the protein kinase that phosphorylates the elF-2 translation initiation factorY Lu, M Wambach, M G Katze, et al.
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