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Nature Communications|September 5, 2020
Mitochondrial CaMKII causes adverse metabolic reprogramming and dilated cardiomyopathyElizabeth D Luczak, Yuejin Wu, Jonathan M Granger, et al.Proceedings of the National Academy of Sciences of the United States of America|June 21, 2017
Evaluation of a proposal for reliable low-cost grid power with 100% wind, water, and solarChristopher T M Clack, Staffan A Qvist, Jay Apt, et al.The Journal of Clinical Investigation|November 5, 2020
Oxidized CaMKII and O-GlcNAcylation cause increased atrial fibrillation in diabetic mice by distinct mechanismsOlurotimi O Mesubi, Adam G Rokita, Neha Abrol, et al.Science Translational Medicine|June 21, 2023
An improved reporter identifies ruxolitinib as a potent and cardioprotective CaMKII inhibitorOscar E Reyes Gaido, Nikoleta Pavlaki, Jonathan M Granger, et al.Nature Communications|May 27, 2021
CaMKII oxidation is a critical performance/disease trade-off acquired at the dawn of vertebrate evolutionQinchuan Wang, Erick O Hernández-Ochoa, Meera C Viswanathan, et al.The Journal of Clinical Investigation|August 5, 2020
MICAL1 constrains cardiac stress responses and protects against disease by oxidizing CaMKIIKlitos Konstantinidis, Vassilios J Bezzerides, Lo Lai, et al.Pageof 16