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American Journal of Physiology. Gastrointestinal and Liver Physiology|October 1, 2011
ATP: a mediator for HCl-induced TRPV1 activation in esophageal mucosaJie Ma, Annamaria Altomare, Florian Rieder, et al.Astrobiology|May 4, 2019
An Integrated Approach for Delivering Current Astrobiology Research to the General PublicErika M Harnett, Diana Johns, Jennifer Gardner, et al.Lupus|June 19, 2015
Drug-like analogues of the parasitic worm-derived immunomodulator ES-62 are therapeutic in the MRL/Lpr model of systemic lupus erythematosusD T Rodgers, M A Pineda, C J Suckling, et al.American Journal of Physiology. Gastrointestinal and Liver Physiology|January 22, 2005
H(2)O(2): a mediator of esophagitis-induced damage to calcium-release mechanisms in cat lower esophageal sphincterWeibiao Cao, Karen M Harnett, Ling Cheng, et al.Immunology|November 21, 1998
MAPkinase: a second site of G-protein regulation of B-cell activation via the antigen receptorsM R Deehan, G G Klaus, M J Holman, et al.Cellular Signalling|November 11, 2003
FcgammaRIIb-mediated negative regulation of BCR signalling is associated with the recruitment of the MAPkinase-phosphatase, Pac-1, and the 3'-inositol phosphatase, PTENKirsty S Brown, Derek Blair, Steven D Reid, et al.Current Topics in Medicinal Chemistry|February 18, 2004
The anti-inflammatory potential of the filarial nematode secreted product, ES-62William Harnett, Margaret M Harnett, Bernard P Leung, et al.Journal of Clinical Pharmacology|July 1, 2006
Predictive power of an in vitro system to assess drug interactions of an antimuscarinic medication: a comparison of in vitro and in vivo drug-drug interaction studies of trospium chloride with digoxinB Sandage, L Sabounjian, J Shipley, et al.Blood|November 24, 2001
Functional coupling of FcgammaRI to nicotinamide adenine dinucleotide phosphate (reduced form) oxidative burst and immune complex trafficking requires the activation of phospholipase D1A J Melendez, L Bruetschy, R A Floto, et al.Arthritis & Rheumatology (Hoboken, N.J.)|February 6, 2014
ES-62 protects against collagen-induced arthritis by resetting interleukin-22 toward resolution of inflammation in the jointsMiguel A Pineda, David T Rodgers, Lamyaa Al-Riyami, et al.Pageof 22