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M Helen Rajpar

Showing results (1-10 of 8) with videos related to

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The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society|August 4, 2012
Hypertrophic chondrocytes have a limited capacity to cope with increases in endoplasmic reticulum stress without triggering the unfolded protein responseLouise H W Kung, M Helen Rajpar, Michael D Briggs, et al.
Plos One|February 19, 2015
Increased classical endoplasmic reticulum stress is sufficient to reduce chondrocyte proliferation rate in the growth plate and decrease bone growthLouise H W Kung, M Helen Rajpar, Richard Preziosi, et al.
Human Molecular Genetics|October 2, 2002
Mutation of the signal peptide region of the bicistronic gene DSPP affects translocation to the endoplasmic reticulum and results in defective dentine biomineralizationM Helen Rajpar, Martin J Koch, Robin M Davies, et al.
Disease Models & Mechanisms|September 19, 2013
A novel transgenic mouse model of growth plate dysplasia reveals that decreased chondrocyte proliferation due to chronic ER stress is a key factor in reduced bone growthBenedetta Gualeni, M Helen Rajpar, Aaron Kellogg, et al.
Plos One|September 22, 2011
Transcriptional profiling of chondrodysplasia growth plate cartilage reveals adaptive ER-stress networks that allow survival but disrupt hypertrophyTrevor L Cameron, Katrina M Bell, Liliana Tatarczuch, et al.
Cell Stress & Chaperones|April 30, 2010
An unfolded protein response is the initial cellular response to the expression of mutant matrilin-3 in a mouse model of multiple epiphyseal dysplasiaSeema Nundlall, M Helen Rajpar, Peter A Bell, et al.
Plos Genetics|October 17, 2009
Targeted induction of endoplasmic reticulum stress induces cartilage pathologyM Helen Rajpar, Ben McDermott, Louise Kung, et al.
Bone Research|June 15, 2026
Murine model of high bone mass osteogenesis imperfecta exhibits bone matrix hyper-mineralization, misaligned mineral crystals, and altered osteoblast differentiationAileen M Barnes, M Helen Rajpar, Joseph E Perosky, et al.
Pageof 1

Showing results (1-10 of 8) with videos related to

Sort By:
Pageof 1
The Journal of Histochemistry and Cytochemistry : Official Journal of the Histochemistry Society|August 4, 2012
Hypertrophic chondrocytes have a limited capacity to cope with increases in endoplasmic reticulum stress without triggering the unfolded protein responseLouise H W Kung, M Helen Rajpar, Michael D Briggs, et al.
Plos One|February 19, 2015
Increased classical endoplasmic reticulum stress is sufficient to reduce chondrocyte proliferation rate in the growth plate and decrease bone growthLouise H W Kung, M Helen Rajpar, Richard Preziosi, et al.
Human Molecular Genetics|October 2, 2002
Mutation of the signal peptide region of the bicistronic gene DSPP affects translocation to the endoplasmic reticulum and results in defective dentine biomineralizationM Helen Rajpar, Martin J Koch, Robin M Davies, et al.
Disease Models & Mechanisms|September 19, 2013
A novel transgenic mouse model of growth plate dysplasia reveals that decreased chondrocyte proliferation due to chronic ER stress is a key factor in reduced bone growthBenedetta Gualeni, M Helen Rajpar, Aaron Kellogg, et al.
Plos One|September 22, 2011
Transcriptional profiling of chondrodysplasia growth plate cartilage reveals adaptive ER-stress networks that allow survival but disrupt hypertrophyTrevor L Cameron, Katrina M Bell, Liliana Tatarczuch, et al.
Cell Stress & Chaperones|April 30, 2010
An unfolded protein response is the initial cellular response to the expression of mutant matrilin-3 in a mouse model of multiple epiphyseal dysplasiaSeema Nundlall, M Helen Rajpar, Peter A Bell, et al.
Plos Genetics|October 17, 2009
Targeted induction of endoplasmic reticulum stress induces cartilage pathologyM Helen Rajpar, Ben McDermott, Louise Kung, et al.
Bone Research|June 15, 2026
Murine model of high bone mass osteogenesis imperfecta exhibits bone matrix hyper-mineralization, misaligned mineral crystals, and altered osteoblast differentiationAileen M Barnes, M Helen Rajpar, Joseph E Perosky, et al.
Pageof 1