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Molecular and Cellular Biology|January 29, 2000
Epstein-Barr virus suppresses a G(2)/M checkpoint activated by genotoxinsM Wade, M J AlldayThe Journal of General Virology|July 1, 1985
Epstein-Barr virus nuclear antigen (EBNA): size polymorphism of EBNA 1M J Allday, A J MacGillivrayThe Journal of General Virology|May 17, 2000
Resistance to TGF-beta1 correlates with a reduction of TGF-beta type II receptor expression in Burkitt's lymphoma and Epstein-Barr virus-transformed B lymphoblastoid cell linesG J Inman, M J AlldayLancet (London, England)|April 16, 1988
Role of epithelium in EBV persistence and pathogenesis of B-cell tumoursM J Allday, D H CrawfordJournal of Immunology (Baltimore, Md. : 1950)|August 18, 2000
Apoptosis induced by TGF-beta 1 in Burkitt's lymphoma cells is caspase 8 dependent but is death receptor independentG J Inman, M J AlldayJournal of Virology|June 1, 1994
Epstein-Barr virus nuclear antigen EBNA3C/6 expression maintains the level of latent membrane protein 1 in G1-arrested cellsM J Allday, P J FarrellThe Journal of General Virology|March 1, 1990
Immortalization of Epstein-Barr virus-infected CD23-negative B lymphocytes by the addition of B cell growth factorT Azim, M J Allday, D H CrawfordThe EMBO Journal|September 1, 1989
EBV gene expression in an NPC-related tumourM M Hitt, M J Allday, T Hara, et al.International Journal of Cancer|March 15, 1990
Deregulated c-myc expression in Epstein-Barr-virus-immortalized B-cells induces altered growth properties and surface phenotype but not tumorigenicityN A Hotchin, M J Allday, D H CrawfordOncogene|March 4, 2000
Epstein-Barr virus EBNA3C can disrupt multiple cell cycle checkpoints and induce nuclear division divorced from cytokinesisG A Parker, R Touitou, M J AlldayPageof 21