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Proceedings of the National Academy of Sciences of the United States of America|April 16, 1996
Suppression of apoptosis by basement membrane requires three-dimensional tissue organization and withdrawal from the cell cycleN Boudreau, Z Werb, M J Bissell
Matrix Biology : Journal of the International Society for Matrix Biology|December 22, 1999
Transcriptional regulation of stromelysin-1 gene expression is altered during progression of mouse mammary epithelial cells from functionally normal to malignantA Lochter, Z Werb, M J Bissell
The Journal of Cell Biology|September 1, 1992
Coordinated expression of extracellular matrix-degrading proteinases and their inhibitors regulates mammary epithelial function during involutionR S Talhouk, M J Bissell, Z Werb
Seminars in Cancer Biology|June 1, 1995
Mammary gland tumor formation in transgenic mice overexpressing stromelysin-1C J Sympson, M J Bissell, Z Werb
Oncogene|March 14, 2000
The matrix metalloproteinase stromelysin-1 acts as a natural mammary tumor promoterM D Sternlicht, M J Bissell, Z Werb
Molecular Biology of the Cell|February 9, 1999
alpha1 and alpha2 integrins mediate invasive activity of mouse mammary carcinoma cells through regulation of stromelysin-1 expressionA Lochter, M Navre, Z Werb, et al.
Annals of the New York Academy of Sciences|January 26, 1999
The significance of matrix metalloproteinases during early stages of tumor progressionA Lochter, M D Sternlicht, Z Werb, et al.
Science (New York, N.Y.)|February 10, 1995
Suppression of ICE and apoptosis in mammary epithelial cells by extracellular matrixN Boudreau, C J Sympson, Z Werb, et al.
The Journal of Cell Biology|December 1, 1996
Rescue of mammary epithelial cell apoptosis and entactin degradation by a tissue inhibitor of metalloproteinases-1 transgeneC M Alexander, E W Howard, M J Bissell, et al.
In Vitro Cellular & Developmental Biology : Journal of the Tissue Culture Association|April 1, 1987
Collagen-fibronectin interactions in normal and Rous sarcoma virus-transformed avian tendon cells: possible mechanisms for increased extracellular matrix turnover after transformationL M Chan, C Hatier, G Parry, et al.
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