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M J Quon

Showing results (11-20 of 64) with videos related to

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The American Journal of Physiology|December 9, 1998
Overestimation of minimal model glucose effectiveness in presence of insulin response is due to undermodelingC Cobelli, F Bettini, A Caumo, et al.
The Journal of Biological Chemistry|December 1, 1995
The mouse obese gene. Genomic organization, promoter activity, and activation by CCAAT/enhancer-binding protein alphaY He, H Chen, M J Quon, et al.
Trends in Endocrinology and Metabolism: TEM|November 1, 1994
Insulin signal transduction pathwaysM J Quon, A J Butte, S I Taylor
Endocrinology|June 21, 2001
Tyr(612) and Tyr(632) in human insulin receptor substrate-1 are important for full activation of insulin-stimulated phosphatidylinositol 3-kinase activity and translocation of GLUT4 in adipose cellsD L Esposito, Y Li, A Cama, et al.
Molecular Endocrinology (Baltimore, Md.)|October 2, 2001
Phosphorylation of PTP1B at Ser(50) by Akt impairs its ability to dephosphorylate the insulin receptorL V Ravichandran, H Chen, Y Li, et al.
The Journal of Biological Chemistry|June 13, 2001
Insulin-stimulated activation of eNOS is independent of Ca2+ but requires phosphorylation by Akt at Ser(1179)M Montagnani, H Chen, V A Barr, et al.
Diabetes|July 1, 1994
Non-insulin-mediated glucose disappearance in subjects with IDDM. Discordance between experimental results and minimal model analysisM J Quon, C Cochran, S I Taylor, et al.
The Journal of Biological Chemistry|November 7, 2000
Protein kinase C-zeta phosphorylates insulin receptor substrate-1 and impairs its ability to activate phosphatidylinositol 3-kinase in response to insulinL V Ravichandran, D L Esposito, J Chen, et al.
Diabetes Research (Edinburgh, Scotland)|January 1, 1994
Direct comparison of standard and insulin modified protocols for minimal model estimation of insulin sensitivity in normal subjectsM J Quon, C Cochran, S I Taylor, et al.
Endocrinology|March 10, 1998
Regulation of leptin promoter function by Sp1, C/EBP, and a novel factorM M Mason, Y He, H Chen, et al.
Pageof 7

Showing results (11-20 of 64) with videos related to

Sort By:
Pageof 7
The American Journal of Physiology|December 9, 1998
Overestimation of minimal model glucose effectiveness in presence of insulin response is due to undermodelingC Cobelli, F Bettini, A Caumo, et al.
The Journal of Biological Chemistry|December 1, 1995
The mouse obese gene. Genomic organization, promoter activity, and activation by CCAAT/enhancer-binding protein alphaY He, H Chen, M J Quon, et al.
Trends in Endocrinology and Metabolism: TEM|November 1, 1994
Insulin signal transduction pathwaysM J Quon, A J Butte, S I Taylor
Endocrinology|June 21, 2001
Tyr(612) and Tyr(632) in human insulin receptor substrate-1 are important for full activation of insulin-stimulated phosphatidylinositol 3-kinase activity and translocation of GLUT4 in adipose cellsD L Esposito, Y Li, A Cama, et al.
Molecular Endocrinology (Baltimore, Md.)|October 2, 2001
Phosphorylation of PTP1B at Ser(50) by Akt impairs its ability to dephosphorylate the insulin receptorL V Ravichandran, H Chen, Y Li, et al.
The Journal of Biological Chemistry|June 13, 2001
Insulin-stimulated activation of eNOS is independent of Ca2+ but requires phosphorylation by Akt at Ser(1179)M Montagnani, H Chen, V A Barr, et al.
Diabetes|July 1, 1994
Non-insulin-mediated glucose disappearance in subjects with IDDM. Discordance between experimental results and minimal model analysisM J Quon, C Cochran, S I Taylor, et al.
The Journal of Biological Chemistry|November 7, 2000
Protein kinase C-zeta phosphorylates insulin receptor substrate-1 and impairs its ability to activate phosphatidylinositol 3-kinase in response to insulinL V Ravichandran, D L Esposito, J Chen, et al.
Diabetes Research (Edinburgh, Scotland)|January 1, 1994
Direct comparison of standard and insulin modified protocols for minimal model estimation of insulin sensitivity in normal subjectsM J Quon, C Cochran, S I Taylor, et al.
Endocrinology|March 10, 1998
Regulation of leptin promoter function by Sp1, C/EBP, and a novel factorM M Mason, Y He, H Chen, et al.
Pageof 7