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M Jungck

Showing results (1-10 of 13) with videos related to

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Zentralblatt Fur Chirurgie|August 10, 2000
[Hereditary carcinoma: pathogenesis and diagnosis]M Jungck
Deutsche Medizinische Wochenschrift (1946)|May 3, 2002
[Cost-effectiveness of screening in familial adenomatous polyposis]M Jungck, T Sauerbruch
Der Internist|July 17, 1999
[Hereditary gastrointestinal tumors]M Jungck, W Friedl, P Propping
Deutsche Medizinische Wochenschrift (1946)|March 17, 2000
[Therapy and after-care of familial adenomatous polyposis and hereditary colorectal carcinoma without polyposis]C Lamberti, M Jungck, R Caspari, et al.
Deutsche Medizinische Wochenschrift (1946)|March 4, 2000
[The diagnosis of hereditary colorectal carcinomas]R Caspari, M Jungck, C Lamberti, et al.
International Journal of Colorectal Disease|February 27, 2004
E-cadherin expression is homogeneously reduced in adenoma from patients with familial adenomatous polyposis: an immunohistochemical study of E-cadherin, beta-catenin and cyclooxygenase-2 expressionM Jungck, F Grünhage, U Spengler, et al.
Gut|May 14, 1999
Microsatellite instability-a useful diagnostic tool to select patients at high risk for hereditary non-polyposis colorectal cancer: a study in different groups of patients with colorectal cancerC Lamberti, R Kruse, C Ruelfs, et al.
Genes, Chromosomes & Cancer|July 9, 1999
Frequent 4-bp deletion in exon 9 of the SMAD4/MADH4 gene in familial juvenile polyposis patientsW Friedl, R Kruse, S Uhlhaas, et al.
Gut|March 15, 2001
Can APC mutation analysis contribute to therapeutic decisions in familial adenomatous polyposis? Experience from 680 FAP familiesW Friedl, R Caspari, M Sengteller, et al.
American Journal of Human Genetics|June 23, 1998
Muir-Torre phenotype has a frequency of DNA mismatch-repair-gene mutations similar to that in hereditary nonpolyposis colorectal cancer families defined by the Amsterdam criteriaR Kruse, A Rütten, C Lamberti, et al.
Pageof 2

Showing results (1-10 of 13) with videos related to

Sort By:
Pageof 2
Zentralblatt Fur Chirurgie|August 10, 2000
[Hereditary carcinoma: pathogenesis and diagnosis]M Jungck
Deutsche Medizinische Wochenschrift (1946)|May 3, 2002
[Cost-effectiveness of screening in familial adenomatous polyposis]M Jungck, T Sauerbruch
Der Internist|July 17, 1999
[Hereditary gastrointestinal tumors]M Jungck, W Friedl, P Propping
Deutsche Medizinische Wochenschrift (1946)|March 17, 2000
[Therapy and after-care of familial adenomatous polyposis and hereditary colorectal carcinoma without polyposis]C Lamberti, M Jungck, R Caspari, et al.
Deutsche Medizinische Wochenschrift (1946)|March 4, 2000
[The diagnosis of hereditary colorectal carcinomas]R Caspari, M Jungck, C Lamberti, et al.
International Journal of Colorectal Disease|February 27, 2004
E-cadherin expression is homogeneously reduced in adenoma from patients with familial adenomatous polyposis: an immunohistochemical study of E-cadherin, beta-catenin and cyclooxygenase-2 expressionM Jungck, F Grünhage, U Spengler, et al.
Gut|May 14, 1999
Microsatellite instability-a useful diagnostic tool to select patients at high risk for hereditary non-polyposis colorectal cancer: a study in different groups of patients with colorectal cancerC Lamberti, R Kruse, C Ruelfs, et al.
Genes, Chromosomes & Cancer|July 9, 1999
Frequent 4-bp deletion in exon 9 of the SMAD4/MADH4 gene in familial juvenile polyposis patientsW Friedl, R Kruse, S Uhlhaas, et al.
Gut|March 15, 2001
Can APC mutation analysis contribute to therapeutic decisions in familial adenomatous polyposis? Experience from 680 FAP familiesW Friedl, R Caspari, M Sengteller, et al.
American Journal of Human Genetics|June 23, 1998
Muir-Torre phenotype has a frequency of DNA mismatch-repair-gene mutations similar to that in hereditary nonpolyposis colorectal cancer families defined by the Amsterdam criteriaR Kruse, A Rütten, C Lamberti, et al.
Pageof 2