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Journal of Immunology (Baltimore, Md. : 1950)|June 8, 2001
MHC class II-regulated central nervous system autoaggression and T cell responses in peripheral lymphoid tissues are dissociated in myelin oligodendrocyte glycoprotein-induced experimental autoimmune encephalomyelitisR Weissert, K L de Graaf, M K Storch, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|August 7, 2001
Acute neuronal apoptosis in a rat model of multiple sclerosisR Meyer, R Weissert, R Diem, et al.
The Journal of Clinical Investigation|September 17, 1998
MHC haplotype-dependent regulation of MOG-induced EAE in ratsR Weissert, E Wallström, M K Storch, et al.
The American Journal of Pathology|July 6, 2000
Multiple sclerosis and chronic autoimmune encephalomyelitis: a comparative quantitative study of axonal injury in active, inactive, and remyelinated lesionsB Kornek, M K Storch, R Weissert, et al.
Brain Pathology (Zurich, Switzerland)|November 6, 1998
Autoimmunity to myelin oligodendrocyte glycoprotein in rats mimics the spectrum of multiple sclerosis pathologyM K Storch, A Stefferl, U Brehm, et al.
Experimental Neurology|June 28, 2012
Re-expression of N-cadherin in remyelinating lesions of experimental inflammatory demyelinationS Hochmeister, M Romauch, J Bauer, et al.
The Journal of Experimental Medicine|June 6, 1998
Vaccination with DNA encoding an immunodominant myelin basic protein peptide targeted to Fc of immunoglobulin G suppresses experimental autoimmune encephalomyelitisA Lobell, R Weissert, M K Storch, et al.
Neuroscience|April 1, 1996
Co-localization of secretoneurin immunoreactivity and macrophage infiltration in the lesions of experimental autoimmune encephalomyelitisM K Storch, R Fischer-Colbrie, T Smith, et al.
Endocrinology|July 19, 2001
Disease progression in chronic relapsing experimental allergic encephalomyelitis is associated with reduced inflammation-driven production of corticosteroneA Stefferl, M K Storch, C Linington, et al.
Brain : a Journal of Neurology|May 17, 2001
Distribution of a calcium channel subunit in dystrophic axons in multiple sclerosis and experimental autoimmune encephalomyelitisB Kornek, M K Storch, J Bauer, et al.
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