M Kalafatis|P Simioni|F Bernardi - JoVE Visualize

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Arteriosclerosis, Thrombosis, and Vascular Biology|February 12, 1999

Phenotype and genotype expression in pseudohomozygous factor VLEIDEN : the need for phenotype analysisM Kalafatis, F Bernardi, P Simioni, et al.

Blood|August 15, 2000

Combinations of 4 mutations (FV R506Q, FV H1299R, FV Y1702C, PT 20210G/A) affecting the prothrombinase complex in a thrombophilic familyE Castoldi, P Simioni, M Kalafatis, et al.

The Journal of Biological Chemistry|September 20, 1996

An antifibrinolytic mechanism describing the prothrombotic effect associated with factor VLeidenL Bajzar, M Kalafatis, P Simioni, et al.

Thrombosis and Haemostasis|October 6, 1998

Molecular bases of pseudo-homozygous APC resistance: the compound heterozygosity for FV R506Q and a FV null mutation results in the exclusive presence of FV Leiden molecules in plasmaE Castoldi, M Kalafatis, B Lunghi, et al.

Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|March 27, 2002

A matrix-assisted laser desorption/ionization time-of-flight based method for screening the 1691G --> A mutation in the factor V geneK Hung, X Sun, H Ding, et al.

Blood|May 16, 1998

Platelet-derived factor Va/Va Leiden cofactor activities are sustained on the surface of activated platelets despite the presence of activated protein CR M Camire, M Kalafatis, P Simioni, et al.

The Journal of Biological Chemistry|August 15, 1997

Increased tissue factor-initiated prothrombin activation as a result of the Arg506 --> Gln mutation in factor VLEIDENC van 't Veer, M Kalafatis, R M Bertina, et al.

Journal of Thrombosis and Haemostasis : JTH|March 8, 2005

Megakaryocytes endocytose and subsequently modify human factor V in vivo to form the entire pool of a unique platelet-derived cofactorW R Gould, P Simioni, J R Silveira, et al.

British Journal of Haematology|September 21, 2001

A highly polymorphic microsatellite in the factor V gene is an informative tool for the study of factor V-related disordersE Castoldi, B Lunghi, F Mingozzi, et al.

Thrombosis and Haemostasis|November 1, 2001

Abnormal propeptide processing resulting in the presence of two abnormal species of protein C in plasma: characterization of the dysfunctional protein C Padua3 (protein C(R-1L/propeptide))P Simioni, M Kalafatis, D Tormene, et al.

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Pageof 43

Arteriosclerosis, Thrombosis, and Vascular Biology|February 12, 1999

Phenotype and genotype expression in pseudohomozygous factor VLEIDEN : the need for phenotype analysisM Kalafatis, F Bernardi, P Simioni, et al.

Blood|August 15, 2000

Combinations of 4 mutations (FV R506Q, FV H1299R, FV Y1702C, PT 20210G/A) affecting the prothrombinase complex in a thrombophilic familyE Castoldi, P Simioni, M Kalafatis, et al.

The Journal of Biological Chemistry|September 20, 1996

An antifibrinolytic mechanism describing the prothrombotic effect associated with factor VLeidenL Bajzar, M Kalafatis, P Simioni, et al.

Thrombosis and Haemostasis|October 6, 1998

Molecular bases of pseudo-homozygous APC resistance: the compound heterozygosity for FV R506Q and a FV null mutation results in the exclusive presence of FV Leiden molecules in plasmaE Castoldi, M Kalafatis, B Lunghi, et al.

Blood Coagulation & Fibrinolysis : an International Journal in Haemostasis and Thrombosis|March 27, 2002

A matrix-assisted laser desorption/ionization time-of-flight based method for screening the 1691G --> A mutation in the factor V geneK Hung, X Sun, H Ding, et al.

Blood|May 16, 1998

Platelet-derived factor Va/Va Leiden cofactor activities are sustained on the surface of activated platelets despite the presence of activated protein CR M Camire, M Kalafatis, P Simioni, et al.

The Journal of Biological Chemistry|August 15, 1997

Increased tissue factor-initiated prothrombin activation as a result of the Arg506 --> Gln mutation in factor VLEIDENC van 't Veer, M Kalafatis, R M Bertina, et al.

Journal of Thrombosis and Haemostasis : JTH|March 8, 2005

Megakaryocytes endocytose and subsequently modify human factor V in vivo to form the entire pool of a unique platelet-derived cofactorW R Gould, P Simioni, J R Silveira, et al.

British Journal of Haematology|September 21, 2001

A highly polymorphic microsatellite in the factor V gene is an informative tool for the study of factor V-related disordersE Castoldi, B Lunghi, F Mingozzi, et al.

Thrombosis and Haemostasis|November 1, 2001

Abnormal propeptide processing resulting in the presence of two abnormal species of protein C in plasma: characterization of the dysfunctional protein C Padua3 (protein C(R-1L/propeptide))P Simioni, M Kalafatis, D Tormene, et al.

Pageof 43